Intracellular acidosis potentiates carotid chemoreceptor responses to hypoxia in the absence of CO2-HCO3-.
Iturriaga
R
R
Laboratory of Neurobiology, Catholic University of Chile, Santiago, Chile.
D000085:Acetates / Q000494:pharmacology; D000138:Acidosis / Q000378:metabolism*; D000200:Action Potentials / Q000187:drug effects; D000818:Animals; D001639:Bicarbonates / Q000378:metabolism*; D002245:Carbon Dioxide / Q000502:physiology*; D002344:Carotid Body / Q000378:metabolism*; D002415:Cats; D015687:Cell Hypoxia / Q000502:physiology*; D006863:Hydrogen-Ion Concentration; D007424:Intracellular Fluid / Q000737:chemistry; D008954:Models, Biological; D009924:Organ Culture Techniques; D010100:Oxygen / Q000502:physiology*; D015398:Signal Transduction / Q000502:physiology; D012966:Sodium Cyanide / Q000494:pharmacology
D000085:Acetates; D001639:Bicarbonates; D002245:Carbon Dioxide; D012966:Sodium Cyanide; D010100:Oxygen