[Inflammatory facial paralysis in MRI. An overview].
In inflammatory peripheral facial nerve palsy pathologically intense, linear and smooth enhancement of the distal intrameatal nerve segment can always be observed on T1-w- SE- MR sequences. The other nerve segments often present with a pathological enhancement as well. On T2-w- SE sequences, a thickening of the distal intrameatal nerve segment can be observed. The pathological enhancement persists over weeks and months; even in patients with complete clinical recovery, a persistent enhancement of the distal intrameatal nerve segment can be demonstrated. No correlation can be established between the intensity of the enhancement, the clinical condition and the electrophysiological data on electroneurography. The persistent enhancement of the different nerve segments is due to a long-lasting breakdown of the blood-peripheral nerve-barrier related to the process of degeneration and regeneration of the facial nerve in inflammatory palsy.
Sartoretti-Schefer
S
S
Institut für Neuroradiologie, Universitätsspital Zürich.
Wichmann
W
W
Valavanis
A
A
D003287:Contrast Media; D005154:Facial Nerve / Q000473:pathology* / Q000503:physiopathology; D005158:Facial Paralysis / Q000175:diagnosis* / Q000503:physiopathology; D005500:Follow-Up Studies; D019786:Gadolinium DTPA; D016697:Herpes Zoster Oticus / Q000175:diagnosis / Q000503:physiopathology; D006801:Humans; D008279:Magnetic Resonance Imaging; D009416:Nerve Regeneration / Q000502:physiology; D009942:Organometallic Compounds; D004369:Pentetic Acid / Q000031:analogs & derivatives; D011115:Polyneuropathies / Q000175:diagnosis* / Q000503:physiopathology
D003287:Contrast Media; D009942:Organometallic Compounds; D004369:Pentetic Acid; D019786:Gadolinium DTPA