Reciprocal paracrine interactions between tumour cells and endothelial cells: the 'angiogenesis progression' hypothesis.
Rak
J
J
Cancer Biology Research Division, Sunnybrook Health Science Center, Toronto, Ontario, Canada.
Filmus
J
J
Kerbel
R S
RS
D000818:Animals; D015179:Colorectal Neoplasms / Q000098:blood supply; D018450:Disease Progression; D016228:Endothelial Growth Factors / Q000378:metabolism; D004730:Endothelium, Vascular / Q000503:physiopathology*; D011905:Genes, ras / Q000502:physiology; D006801:Humans; D008222:Lymphokines / Q000378:metabolism; D008545:Melanoma / Q000098:blood supply; D051379:Mice; D009362:Neoplasm Metastasis; D009369:Neoplasms / Q000098:blood supply*; D009389:Neovascularization, Pathologic / Q000503:physiopathology*; D051381:Rats; D012878:Skin Neoplasms / Q000098:blood supply; D042461:Vascular Endothelial Growth Factor A; D042442:Vascular Endothelial Growth Factors
D016228:Endothelial Growth Factors; D008222:Lymphokines; D042461:Vascular Endothelial Growth Factor A; D042442:Vascular Endothelial Growth Factors