Hyperexpression of GAD in the islets may be relevant but is it sufficient to induce autoimmune insulitis?
Pozzilli
P
P
University of Rome La Sapienza, Italy.
Leslie
R D
RD
D001324:Autoantigens / Q000096:biosynthesis / Q000276:immunology; D001327:Autoimmune Diseases / Q000201:enzymology / Q000276:immunology*; D002648:Child; D003922:Diabetes Mellitus, Type 1 / Q000201:enzymology* / Q000276:immunology* / Q000517:prevention & control; D004200:Diseases in Twins; D015870:Gene Expression; D005968:Glutamate Decarboxylase / Q000096:biosynthesis* / Q000276:immunology; D006801:Humans; D007515:Islets of Langerhans / Q000201:enzymology* / Q000276:immunology*; D010182:Pancreatic Diseases / Q000201:enzymology / Q000276:immunology*
D001324:Autoantigens; D005968:Glutamate Decarboxylase