Expression of a negative dominant mutant of human p55 tumor necrosis factor-receptor inhibits TNF and monocyte-induced activation in porcine aortic endothelial cells.
Stroka
D M
DM
Sandoz Center for Immunobiology, Deaconess Hospital, Harvard Medical School, Boston, MA 02215, USA.
Cooper
J T
JT
Brostjan
C
C
Millan
M T
MT
Goodman
D J
DJ
Wrighton
C J
CJ
Bach
F H
FH
Ferran
C
C
D000818:Animals; D015703:Antigens, CD / Q000096:biosynthesis* / Q000235:genetics / Q000502:physiology; D001011:Aorta; D002478:Cells, Cultured; D004730:Endothelium, Vascular / Q000187:drug effects / Q000502:physiology*; D005799:Genes, Dominant; D006085:Graft Survival; D006801:Humans; D009000:Monocytes / Q000502:physiology*; D018124:Receptors, Tumor Necrosis Factor / Q000096:biosynthesis* / Q000235:genetics / Q000502:physiology; D047888:Receptors, Tumor Necrosis Factor, Type I; D011994:Recombinant Proteins / Q000096:biosynthesis; D013552:Swine; D014162:Transfection; D014183:Transplantation, Heterologous; D014409:Tumor Necrosis Factor-alpha / Q000494:pharmacology*
D015703:Antigens, CD; D018124:Receptors, Tumor Necrosis Factor; D047888:Receptors, Tumor Necrosis Factor, Type I; D011994:Recombinant Proteins; D014409:Tumor Necrosis Factor-alpha