title: Ether lipid metabolism, GPI anchor biosynthesis, and signal transduction are putative targets for anti-leishmanial alkyl phospholipid analogues.
authors.affiliation: King's College London, United Kingdom.
authors.forename: H
authors.initials: H
authors.lastname: Lux
authors.forename: D T
authors.initials: DT
authors.lastname: Hart
authors.forename: P J
authors.initials: PJ
authors.lastname: Parker
authors.forename: T
authors.initials: T
authors.lastname: Klenner
mesh_terms: D000818:Animals; D000981:Antiprotozoal Agents / Q000494:pharmacology*; D017261:Glycosylphosphatidylinositols / Q000096:biosynthesis*; D007527:Isoenzymes / Q000037:antagonists & inhibitors / Q000378:metabolism; D007894:Leishmania mexicana / Q000187:drug effects* / Q000201:enzymology / Q000378:metabolism; D007928:Lethal Dose 50; D050356:Lipid Metabolism; D010743:Phospholipids / Q000494:pharmacology*; D011493:Protein Kinase C / Q000037:antagonists & inhibitors / Q000378:metabolism; D015398:Signal Transduction / Q000187:drug effects*
chemical_list: D000981:Antiprotozoal Agents; D017261:Glycosylphosphatidylinositols; D007527:Isoenzymes; D010743:Phospholipids; D011493:Protein Kinase C