Myocardial injury after electrical therapy for cardiac arrhythmias assessed by troponin-T release.
Episodes of ventricular fibrillation with subsequent intracardiac, and to a lesser extent, external defibrillation give rise to a statistically significant increase in S-troponin T, S-CK-MB(mass) and S-myoglobin indicative of a minor myocardial injury or dysfunction. In contrast, no such signs were observed after external direct-current conversion of atrial fibrillation using high energies, or after pace-terminated ventricular tachycardia.
Runsiö
M
M
Department of Cardiothoracic Surgery, The Karolinska Institute at the Karolinska Hospital, Stockholm, Sweden.
Kallner
A
A
Källner
G
G
Rosenqvist
M
M
Bergfeldt
L
L
D000328:Adult; D000368:Aged; D019540:Area Under Curve; D001145:Arrhythmias, Cardiac / Q000097:blood / Q000628:therapy*; D015415:Biomarkers / Q000097:blood; D003402:Creatine Kinase / Q000097:blood; D004599:Electric Stimulation Therapy / Q000009:adverse effects*; D005260:Female; D006335:Heart Injuries / Q000097:blood / Q000209:etiology*; D006801:Humans; D007527:Isoenzymes; D008297:Male; D008875:Middle Aged; D009206:Myocardium / Q000378:metabolism*; D009211:Myoglobin / Q000097:blood; D014336:Troponin / Q000097:blood*; D020107:Troponin T
D015415:Biomarkers; D007527:Isoenzymes; D009211:Myoglobin; D014336:Troponin; D020107:Troponin T; D003402:Creatine Kinase