Acute stress impairs lymphocyte function but spares pancreatic endocrine function.
In the same animal, following HS and recovery, pancreatic islet function remains intact while immunologic functions are impaired. Cellular responses to thermal stresses are complex and tissue specific.
Rewinski
M J
MJ
Department of Surgery, Hartford Hospital, Connecticut 06102-5037, USA.
Krastins
M
M
Schweizer
R T
RT
Perdrizet
G A
GA
D000818:Animals; D001786:Blood Glucose / Q000378:metabolism; D002478:Cells, Cultured; D005947:Glucose / Q000494:pharmacology; D005951:Glucose Tolerance Test; D006358:Hot Temperature; D007328:Insulin / Q000378:metabolism*; D000078790:Insulin Secretion; D007376:Interleukin-2 / Q000096:biosynthesis; D007515:Islets of Langerhans / Q000187:drug effects / Q000378:metabolism*; D008213:Lymphocyte Activation; D008214:Lymphocytes / Q000276:immunology*; D051381:Rats; D011917:Rats, Inbred Lew; D012016:Reference Values; D013154:Spleen / Q000276:immunology; D013312:Stress, Physiological; D013806:Theophylline / Q000494:pharmacology; D013997:Time Factors
D001786:Blood Glucose; D007328:Insulin; D007376:Interleukin-2; D013806:Theophylline; D005947:Glucose