N-methyl-D-aspartate receptor-mediated events contribute to neurovascular breakdown during experimental allergic encephalomyelitis.
Paul
C
C
School of Pharmacy and Pharmacology, University of Bath, UK.
Scott
G S
GS
Barbour
M J
MJ
Seiler
N
N
Bolton
C
C
D000818:Animals; D001812:Blood-Brain Barrier; D002199:Capillary Permeability; D002531:Cerebellum / Q000098:blood supply* / Q000378:metabolism; D016291:Dizocilpine Maleate / Q000494:pharmacology; D004681:Encephalomyelitis, Autoimmune, Experimental / Q000503:physiopathology*; D008297:Male; D010880:Piperidines / Q000494:pharmacology; D011700:Putrescine / Q000134:cerebrospinal fluid / Q000378:metabolism; D051381:Rats; D011917:Rats, Inbred Lew; D016194:Receptors, N-Methyl-D-Aspartate / Q000502:physiology*; D013116:Spinal Cord / Q000098:blood supply* / Q000378:metabolism; D014665:Vasodilator Agents / Q000494:pharmacology
D010880:Piperidines; D016194:Receptors, N-Methyl-D-Aspartate; D014665:Vasodilator Agents; D016291:Dizocilpine Maleate; C010739:ifenprodil; D011700:Putrescine