Glucocorticoid inhibition of cytokine-induced E-selectin promoter activation.
Ray
K P
KP
Cell Biology Unit, Glaxo Wellcome Research, Stevenage, Herts, UK.
Searle
N
N
D000730:Androstadienes / Q000494:pharmacology; D019819:Budesonide; D002460:Cell Line; D003907:Dexamethasone / Q000494:pharmacology*; D019040:E-Selectin / Q000096:biosynthesis* / Q000235:genetics; D000068298:Fluticasone; D017930:Genes, Reporter; D005938:Glucocorticoids / Q000494:pharmacology*; D006801:Humans; D007375:Interleukin-1 / Q000037:antagonists & inhibitors / Q000494:pharmacology*; D007700:Kinetics; D008168:Lung; D016328:NF-kappa B / Q000378:metabolism*; D011282:Pregnenediones / Q000494:pharmacology; D011401:Promoter Regions, Genetic; D011993:Recombinant Fusion Proteins / Q000096:biosynthesis; D014162:Transfection; D014409:Tumor Necrosis Factor-alpha / Q000037:antagonists & inhibitors / Q000494:pharmacology*
D000730:Androstadienes; D019040:E-Selectin; D005938:Glucocorticoids; D007375:Interleukin-1; D016328:NF-kappa B; D011282:Pregnenediones; D011993:Recombinant Fusion Proteins; D014409:Tumor Necrosis Factor-alpha; D019819:Budesonide; D003907:Dexamethasone; D000068298:Fluticasone