Functional interaction between Na(+)-K+ pump and KATP channels in the CNS neurons under experimental brain ischemia.
Akaike
N
N
Department of Physiology, Faculty of Medicine, Kyushu University, Fukuoka, Japan.
Jin
Y H
YH
Koyama
S
S
D000255:Adenosine Triphosphate / Q000502:physiology*; D000818:Animals; D002545:Brain Ischemia / Q000378:metabolism* / Q000473:pathology; D002118:Calcium / Q000378:metabolism; D003600:Cytosol / Q000378:metabolism; D004553:Electric Conductivity; D018690:Excitatory Amino Acid Agonists / Q000494:pharmacology; D007003:Hypoglycemia / Q000378:metabolism / Q000473:pathology; D000860:Hypoxia / Q000378:metabolism / Q000473:pathology; D008125:Locus Coeruleus / Q000187:drug effects / Q000378:metabolism* / Q000473:pathology; D016202:N-Methylaspartate / Q000494:pharmacology; D009474:Neurons / Q000187:drug effects / Q000378:metabolism* / Q000502:physiology; D009994:Osmolar Concentration; D015221:Potassium Channels / Q000378:metabolism*; D051381:Rats; D000254:Sodium-Potassium-Exchanging ATPase / Q000502:physiology*
D018690:Excitatory Amino Acid Agonists; D015221:Potassium Channels; D016202:N-Methylaspartate; D000255:Adenosine Triphosphate; D000254:Sodium-Potassium-Exchanging ATPase; D002118:Calcium