Plasmodium falciparum: soluble thrombospondin increases cytoadherence of parasitized erythrocytes to human microvascular endothelium under shear flow conditions.
Siano
J P
JP
School of Chemical Engineering, Georgia Institute of Technology, Atlanta 30332-0100, USA.
Grady
K K
KK
Millet
P
P
Swerlick
R A
RA
Wick
T M
TM
D000704:Analysis of Variance; D000818:Animals; D018955:CD36 Antigens / Q000378:metabolism; D002448:Cell Adhesion; D015815:Cell Adhesion Molecules / Q000097:blood* / Q000737:chemistry / Q000502:physiology; D002478:Cells, Cultured; D016895:Culture Media, Serum-Free; D004730:Endothelium, Vascular / Q000166:cytology / Q000378:metabolism*; D004912:Erythrocytes / Q000378:metabolism / Q000469:parasitology*; D006801:Humans; D016778:Malaria, Falciparum / Q000097:blood* / Q000209:etiology; D008562:Membrane Glycoproteins / Q000097:blood* / Q000737:chemistry / Q000502:physiology; D008833:Microcirculation; D010963:Plasmodium falciparum / Q000378:metabolism*; D012995:Solubility; D019699:Thrombospondins
D018955:CD36 Antigens; D015815:Cell Adhesion Molecules; D016895:Culture Media, Serum-Free; D008562:Membrane Glycoproteins; D019699:Thrombospondins