Pathogenesis of Clostridium perfringens infection: mechanisms and mediators of shock.
Stevens
D L
DL
Infectious Diseases Section, Veterans Affairs Medical Center, Boise, Idaho 83702, USA.
Bryant
A E
AE
D000818:Animals; D001427:Bacterial Toxins / Q000633:toxicity; D002135:Calcium-Binding Proteins; D003016:Clostridium perfringens / Q000472:pathogenicity*; D005738:Gas Gangrene / Q000209:etiology*; D006460:Hemolysin Proteins; D006801:Humans; D008297:Male; D011817:Rabbits; D012772:Shock, Septic / Q000209:etiology*; D014409:Tumor Necrosis Factor-alpha / Q000096:biosynthesis; D010738:Type C Phospholipases / Q000633:toxicity; D014655:Vascular Resistance
D001427:Bacterial Toxins; D002135:Calcium-Binding Proteins; D006460:Hemolysin Proteins; D014409:Tumor Necrosis Factor-alpha; C020452:Clostridium perfringens theta-toxin; D010738:Type C Phospholipases; C040649:alpha toxin, Clostridium perfringens