Use of transgenic mice to eliminate retinoic acid receptor function in specific tissues.
Saitou
M
M
Department of Cell Biology, Kyoto University Faculty of Medicine, Japan.
Tanaka
T
T
Kakizuka
A
A
D000595:Amino Acid Sequence; D000818:Animals; D002460:Cell Line; D002467:Cell Nucleus / Q000378:metabolism; D002874:Chromosome Mapping; D017124:Conserved Sequence; D006801:Humans; D051379:Mice; D008822:Mice, Transgenic; D008969:Molecular Sequence Data; D018774:Oncogene Proteins v-erbA / Q000737:chemistry; D010641:Phenotype; D018168:Receptors, Retinoic Acid / Q000096:biosynthesis* / Q000737:chemistry / Q000235:genetics*; D011988:Receptors, Thyroid Hormone / Q000096:biosynthesis / Q000737:chemistry / Q000235:genetics*; D011994:Recombinant Proteins / Q000096:biosynthesis / Q000737:chemistry; D016415:Sequence Alignment; D017386:Sequence Homology, Amino Acid; D014158:Transcription, Genetic; D014162:Transfection / Q000379:methods
D018774:Oncogene Proteins v-erbA; D018168:Receptors, Retinoic Acid; D011988:Receptors, Thyroid Hormone; D011994:Recombinant Proteins