Stressor-specific activation of catecholaminergic systems: implications for stress-related hypothalamic-pituitary-adrenocortical responses.
Pacák
K
K
Department of Medicine, National Institute of Neurological Disorders and Stroke, Washington, D.C. USA.
Baffi
J S
JS
Kvetnanský
R
R
Goldstein
D S
DS
Palkovits
M
M
D000324:Adrenocorticotropic Hormone / Q000097:blood / Q000378:metabolism; D000818:Animals; D001933:Brain Stem / Q000378:metabolism; D002395:Catecholamines / Q000502:physiology*; D007030:Hypothalamo-Hypophyseal System / Q000502:physiology / Q000503:physiopathology*; D008297:Male; D009474:Neurons / Q000378:metabolism; D009638:Norepinephrine / Q000378:metabolism; D010286:Paraventricular Hypothalamic Nucleus / Q000378:metabolism; D010913:Pituitary-Adrenal System / Q000502:physiology / Q000503:physiopathology*; D016760:Proto-Oncogene Proteins c-fos / Q000096:biosynthesis; D051381:Rats; D011921:Rats, Inbred WKY; D013312:Stress, Physiological / Q000503:physiopathology*; D013315:Stress, Psychological / Q000503:physiopathology*
D002395:Catecholamines; D016760:Proto-Oncogene Proteins c-fos; D000324:Adrenocorticotropic Hormone; D009638:Norepinephrine