The modulation of corticoaccumbens transmission by limbic afferents and dopamine: a model for the pathophysiology of schizophrenia.
Grace
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AA
Department of Neuroscience, University of Pittsburgh, Pennsylvania 15260, USA.
Moore
H
H
O'Donnell
P
P
D000344:Afferent Pathways / Q000502:physiology / Q000503:physiopathology; D000818:Animals; D002540:Cerebral Cortex / Q000502:physiology / Q000503:physiopathology*; D004298:Dopamine / Q000502:physiology*; D018491:Dopamine Agonists / Q000494:pharmacology; D018492:Dopamine Antagonists / Q000494:pharmacology; D004558:Electric Stimulation; D019706:Excitatory Postsynaptic Potentials / Q000187:drug effects; D006624:Hippocampus / Q000502:physiology / Q000503:physiopathology; D006801:Humans; D066298:In Vitro Techniques; D008032:Limbic System / Q000502:physiology / Q000503:physiopathology*; D008959:Models, Neurological; D009412:Nerve Fibers / Q000502:physiology; D009714:Nucleus Accumbens / Q000502:physiology / Q000503:physiopathology*; D017447:Receptors, Dopamine D1 / Q000502:physiology; D017448:Receptors, Dopamine D2 / Q000502:physiology; D012559:Schizophrenia / Q000503:physiopathology*; D009435:Synaptic Transmission
D018491:Dopamine Agonists; D018492:Dopamine Antagonists; D017447:Receptors, Dopamine D1; D017448:Receptors, Dopamine D2; D004298:Dopamine