Use of transgenic mice to study activation of retinoic acid-responsive promoters.
Wojnowski
L
L
Section on Genetics, National Institute of Mental Health, National Institutes of Health, Bethesda, Maryland 20892, USA.
Zimmer
A
A
D000818:Animals; D001665:Binding Sites; D002460:Cell Line; D019281:Dimerization; D005260:Female; D005333:Fetus; D015971:Gene Expression Regulation, Enzymologic / Q000187:drug effects*; D017930:Genes, Reporter; D007763:Lac Operon; D051379:Mice; D008822:Mice, Transgenic; D011247:Pregnancy; D011401:Promoter Regions, Genetic / Q000187:drug effects*; D018168:Receptors, Retinoic Acid / Q000096:biosynthesis / Q000235:genetics*; D017384:Sequence Deletion; D014162:Transfection; D014212:Tretinoin / Q000494:pharmacology*; D001616:beta-Galactosidase / Q000096:biosynthesis / Q000235:genetics
D018168:Receptors, Retinoic Acid; C082925:retinoic acid receptor beta; D014212:Tretinoin; D001616:beta-Galactosidase