Compensatory long-term effect of perinatal hypoxia-ischemia. A possible mechanism for neuroprotection?
Binienda
Z
Z
Division of Neurotoxicology, Food and Drug Administration, Jefferson, Arkansas 72079-9502, USA. zbinienda@nctr.fda.gov
D000818:Animals; D000831:Animals, Newborn; D001921:Brain / Q000187:drug effects / Q000473:pathology*; D002545:Brain Ischemia / Q000139:chemically induced / Q000473:pathology* / Q000523:psychology*; D003216:Conditioning, Operant; D004734:Energy Metabolism; D005333:Fetus; D002534:Hypoxia, Brain / Q000139:chemically induced / Q000473:pathology* / Q000523:psychology*; D007858:Learning / Q000502:physiology*; D008297:Male; D009042:Motivation; D015999:Multivariate Analysis; D018696:Neuroprotective Agents; D009498:Neurotoxins; D009574:Nitro Compounds; D011422:Propionates / Q000633:toxicity*; D051381:Rats; D017207:Rats, Sprague-Dawley; D013385:Succinate Dehydrogenase / Q000037:antagonists & inhibitors
D018696:Neuroprotective Agents; D009498:Neurotoxins; D009574:Nitro Compounds; D011422:Propionates; D013385:Succinate Dehydrogenase; C015392:3-nitropropionic acid