Functional inactivation of signalling molecules via transdominant negative mutants.
Kitamura
M
M
Department of Medicine, University College London Medical School, UK. m.kitamura@medicine.ucl.ac.uk
Ishikawa
Y
Y
D000818:Animals; D002478:Cells, Cultured; D004268:DNA-Binding Proteins / Q000235:genetics / Q000502:physiology; D005920:Glomerular Mesangium / Q000378:metabolism*; D016296:Mutagenesis; D016328:NF-kappa B / Q000235:genetics / Q000502:physiology; D009687:Nuclear Proteins / Q000235:genetics / Q000502:physiology; D011518:Proto-Oncogene Proteins / Q000235:genetics / Q000378:metabolism; D016755:Proto-Oncogene Proteins c-jun / Q000235:genetics / Q000502:physiology; D051381:Rats; D026362:Serum Response Factor; D015398:Signal Transduction; D018808:Transcription Factor AP-1 / Q000235:genetics / Q000502:physiology; D014157:Transcription Factors / Q000235:genetics* / Q000502:physiology*; D014162:Transfection; D001616:beta-Galactosidase / Q000235:genetics; D050807:ets-Domain Protein Elk-1
D004268:DNA-Binding Proteins; C494114:Elk1 protein, rat; D016328:NF-kappa B; D009687:Nuclear Proteins; D011518:Proto-Oncogene Proteins; D016755:Proto-Oncogene Proteins c-jun; D026362:Serum Response Factor; D018808:Transcription Factor AP-1; D014157:Transcription Factors; D050807:ets-Domain Protein Elk-1; D001616:beta-Galactosidase