Extracellular matrix remodeling in heart failure: a role for de novo angiotensin II generation.
Weber
K T
KT
Department of Internal Medicine, University of Missouri Health Sciences Center, Columbia 65212, USA.
D000368:Aged; D000804:Angiotensin II / Q000096:biosynthesis* / Q000097:blood; D000806:Angiotensin-Converting Enzyme Inhibitors / Q000627:therapeutic use; D000818:Animals; D003094:Collagen / Q000378:metabolism; D005260:Female; D005355:Fibrosis; D006333:Heart Failure / Q000209:etiology / Q000473:pathology* / Q000503:physiopathology*; D006801:Humans; D009203:Myocardial Infarction / Q000150:complications* / Q000473:pathology / Q000503:physiopathology; D009210:Myofibrils; D007703:Peptidyl-Dipeptidase A / Q000187:drug effects / Q000378:metabolism; D018487:Ventricular Dysfunction, Left / Q000209:etiology / Q000473:pathology* / Q000503:physiopathology*
D000806:Angiotensin-Converting Enzyme Inhibitors; D000804:Angiotensin II; D003094:Collagen; D007703:Peptidyl-Dipeptidase A