Neutrophil Fc receptors signal through PI 3-kinase to trigger a respiratory burst.
Lang
M L
ML
Dept Of Molecular & Cellular Pathology, Ninewells Hospital & Medical School, Dundee, Scotland, UK.
Kerr
M A
MA
D002867:Chromones / Q000494:pharmacology; D004791:Enzyme Inhibitors / Q000494:pharmacology; D006801:Humans; D007070:Immunoglobulin A / Q000494:pharmacology; D007074:Immunoglobulin G / Q000494:pharmacology; D066298:In Vitro Techniques; D007700:Kinetics; D008163:Luminescent Measurements; D009025:Morpholines / Q000494:pharmacology; D009504:Neutrophils / Q000201:enzymology / Q000276:immunology / Q000502:physiology*; D019869:Phosphatidylinositol 3-Kinases / Q000097:blood*; D011961:Receptors, Fc / Q000187:drug effects / Q000502:physiology*; D016897:Respiratory Burst / Q000187:drug effects; D015398:Signal Transduction; D013755:Tetradecanoylphorbol Acetate / Q000494:pharmacology
D002867:Chromones; D004791:Enzyme Inhibitors; D007070:Immunoglobulin A; D007074:Immunoglobulin G; D009025:Morpholines; D011961:Receptors, Fc; C085911:2-(4-morpholinyl)-8-phenyl-4H-1-benzopyran-4-one; D019869:Phosphatidylinositol 3-Kinases; D013755:Tetradecanoylphorbol Acetate