Specific-pathogen-free conditions enhance inflammatory bowel disease in T-cell receptor knockout, but not C3H/HeJBir mice.
Gaskins
H R
HR
Division of Nutritional Sciences, University of Illinois at Urbana-Champaign, USA.
Vondrak-Juergens
G L
GL
McCracken
B A
BA
Woolsey
J H
JH
D000818:Animals; D002432:Cecum / Q000473:pathology; D003106:Colon / Q000473:pathology; D004195:Disease Models, Animal; D004198:Disease Susceptibility; D005260:Female; D015994:Incidence; D015212:Inflammatory Bowel Diseases / Q000453:epidemiology / Q000235:genetics / Q000662:veterinary*; D008297:Male; D051379:Mice; D008809:Mice, Inbred C3H / Q000235:genetics*; D018345:Mice, Knockout / Q000235:genetics*; D009154:Mutation; D011948:Receptors, Antigen, T-Cell / Q000235:genetics*; D012376:Rodent Diseases / Q000453:epidemiology / Q000235:genetics* / Q000503:physiopathology; D012720:Severity of Illness Index; D013047:Specific Pathogen-Free Organisms
D011948:Receptors, Antigen, T-Cell