[Development of prediction for NIDDM from a view point of genetics].
Sanke
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First Department of Internal Medicine, Wakayama University of Medical Science.
D000682:Amyloid / Q000235:genetics; D004272:DNA, Mitochondrial / Q000235:genetics; D004268:DNA-Binding Proteins; D003924:Diabetes Mellitus, Type 2 / Q000235:genetics*; D005941:Glucokinase / Q000235:genetics; D006006:Glycogen Synthase / Q000235:genetics; D051537:Hepatocyte Nuclear Factor 1; D051538:Hepatocyte Nuclear Factor 1-alpha; D051539:Hepatocyte Nuclear Factor 1-beta; D006801:Humans; D007328:Insulin / Q000235:genetics; D058228:Islet Amyloid Polypeptide; D009154:Mutation; D009687:Nuclear Proteins; D011972:Receptor, Insulin / Q000235:genetics; D014157:Transcription Factors / Q000235:genetics
D000682:Amyloid; D004272:DNA, Mitochondrial; D004268:DNA-Binding Proteins; C496487:HNF1A protein, human; C496495:HNF1B protein, human; D051538:Hepatocyte Nuclear Factor 1-alpha; D007328:Insulin; D058228:Islet Amyloid Polypeptide; D009687:Nuclear Proteins; D014157:Transcription Factors; D051537:Hepatocyte Nuclear Factor 1; D051539:Hepatocyte Nuclear Factor 1-beta; D006006:Glycogen Synthase; D005941:Glucokinase; D011972:Receptor, Insulin