TAP1, TAP2, LMP2, DMA, and DMB genetic polymorphisms in renal transplantation.
Kobayashi
T
T
Department of Surgery II, Nagoya University School of Medicine, Japan.
Yokoyama
I
I
Hayashi
S
S
Negita
M
M
Namil
Y
Y
Katayama
A
A
Nagasaka
T
T
Koike
C
C
Tachi
Y
Y
Mei
G L
GL
Haba
T
T
Tominaga
Y
Y
Naruse
T
T
Inoko
H
H
Uchida
K
K
Takagi
H
H
D000071181:ATP Binding Cassette Transporter, Subfamily B, Member 2; D000071450:ATP Binding Cassette Transporter, Subfamily B, Member 3; D018528:ATP-Binding Cassette Transporters / Q000235:genetics*; D003546:Cysteine Endopeptidases; D004351:Drug Resistance; D006084:Graft Rejection / Q000235:genetics; D006681:HLA-D Antigens / Q000235:genetics*; D000949:Histocompatibility Antigens Class II; D006801:Humans; D016030:Kidney Transplantation / Q000502:physiology*; D011110:Polymorphism, Genetic / Q000235:genetics*; D011506:Proteins / Q000235:genetics*; D013256:Steroids / Q000627:therapeutic use
D000071181:ATP Binding Cassette Transporter, Subfamily B, Member 2; D000071450:ATP Binding Cassette Transporter, Subfamily B, Member 3; D018528:ATP-Binding Cassette Transporters; C416189:H2-M antigens; D006681:HLA-D Antigens; C084245:HLA-DM antigens; D000949:Histocompatibility Antigens Class II; D011506:Proteins; D013256:Steroids; C073119:TAP1 protein, human; C071313:LMP-2 protein; C071692:TAP2 protein, human; D003546:Cysteine Endopeptidases