Autoimmune and inflammatory responses may have an additive effect in postpercutaneous transluminal coronary angioplasty restenosis.
Patients who had an increase in their serum amyloid type A level of > 100% in the first 24 hours after percutaneous transluminal coronary angioplasty (PTCA) and also developed a positive antibody result (antinuclear factor or anticardiolipin), had a relative risk of 10.6 for developing restenosis in the first year after PTCA.
Blum
A
A
Department of Cardiology, Tel-Aviv Medical Center, Sackler Medical School, Tel-Aviv University, Israel.
Vardinon
N
N
Kaplan
G
G
Laniado
S
S
Yust
I
I
Burk
M
M
Miller
H
H
D000789:Angina, Unstable / Q000276:immunology* / Q000628:therapy; D015906:Angioplasty, Balloon, Coronary; D000917:Antibody Formation; D001323:Autoantibodies; D015551:Autoimmunity; D003251:Constriction, Pathologic; D005260:Female; D006801:Humans; D007249:Inflammation / Q000276:immunology*; D008297:Male; D008875:Middle Aged; D011446:Prospective Studies; D012008:Recurrence; D000685:Serum Amyloid A Protein / Q000032:analysis
D001323:Autoantibodies; D000685:Serum Amyloid A Protein