[Possible role of nitric oxide in pathogenesis of model seizure of the different etiology].
Bashkatova
V G
VG
Vitskova
G Iu
GIu
Narkevich
V B
VB
Mikoian
V D
VD
Vanin
A F
AF
Raevskiĭ
K S
KS
D000818:Animals; D001921:Brain / Q000378:metabolism; D003292:Convulsants / Q000633:toxicity; D004578:Electron Spin Resonance Spectroscopy; D004597:Electroshock; D018690:Excitatory Amino Acid Agonists / Q000633:toxicity; D015227:Lipid Peroxidation; D008297:Male; D016202:N-Methylaspartate / Q000633:toxicity; D009569:Nitric Oxide / Q000378:metabolism*; D010433:Pentylenetetrazole / Q000633:toxicity; D051381:Rats; D017208:Rats, Wistar; D016194:Receptors, N-Methyl-D-Aspartate / Q000819:agonists; D012640:Seizures / Q000139:chemically induced / Q000209:etiology / Q000378:metabolism* / Q000503:physiopathology; D012663:Semicarbazides / Q000633:toxicity; D005680:gamma-Aminobutyric Acid / Q000378:metabolism
D003292:Convulsants; D018690:Excitatory Amino Acid Agonists; D016194:Receptors, N-Methyl-D-Aspartate; D012663:Semicarbazides; D009569:Nitric Oxide; D005680:gamma-Aminobutyric Acid; C005151:thiosemicarbazide; D016202:N-Methylaspartate; D010433:Pentylenetetrazole
Vozmozhnaia rol' oksida azota v patogeneze model'nykh sudorozhnykh sostoianiĭ razlichnoĭ prirody.