Contribution of myocyte apoptosis to myocardial infarction?
Bartling
B
B
Institute für Pathophysiologie, Martin-Luther-Universität Halle-Wittenberg, Halle/Saale.
Holtz
J
J
Darmer
D
D
D000818:Animals; D017209:Apoptosis; D019254:Genes, bcl-2 / Q000235:genetics / Q000502:physiology; D007334:Insulin-Like Growth Factor I / Q000502:physiology; D051379:Mice; D008928:Mitochondria / Q000473:pathology; D009203:Myocardial Infarction / Q000235:genetics / Q000473:pathology*; D017202:Myocardial Ischemia / Q000473:pathology; D009206:Myocardium / Q000473:pathology*; D009336:Necrosis; D011518:Proto-Oncogene Proteins / Q000502:physiology; D019253:Proto-Oncogene Proteins c-bcl-2 / Q000502:physiology; D011817:Rabbits; D051381:Rats; D051028:bcl-2-Associated X Protein; D051020:bcl-X Protein
C494812:Bcl2l1 protein, mouse; C494813:Bcl2l1 protein, rat; D011518:Proto-Oncogene Proteins; D019253:Proto-Oncogene Proteins c-bcl-2; D051028:bcl-2-Associated X Protein; D051020:bcl-X Protein; D007334:Insulin-Like Growth Factor I