[Hepatotoxic encephalopathy versus Leigh syndrome--a case report].
Low perfusion of the liver due to hypovolaemia and sepsis-induced pathological distribution of blood volume can lead to severe liver disturbances. Damage to the liver as shock organ is manifold and affects other functions. Increased serum levels of ammonia and zerebral symptoms with disturbances of neurotransmission are responsible for the development of encephalopathia. Based on a case report, the differential diagnosis of Leigh-Syndrome as a mitochondric encephalopathy with uniform morphologic form is discussed. Long lasting parenteral nutrition, sepsis, metabolic imbalance and disturbance of the electrolyte balance can influence the extent of the mitochondric encephalopathy.
Dressler
C
C
Krankenhausbetrieb von Berlin Friedrichshain, Abteilung für Anästhesie und Intensivtherapie.
Kirowa-Sunkel
R
R
Gründler
M
M
D001284:Atrophy; D002552:Cerebral Ventricles / Q000473:pathology; D002648:Child; D003937:Diagnosis, Differential; D005260:Female; D005625:Frontal Lobe / Q000473:pathology; D006501:Hepatic Encephalopathy / Q000175:diagnosis*; D006801:Humans; D007415:Intestinal Obstruction / Q000601:surgery*; D007416:Intestinal Perforation / Q000601:surgery*; D007888:Leigh Disease / Q000175:diagnosis*; D008279:Magnetic Resonance Imaging; D011183:Postoperative Complications / Q000175:diagnosis*; D014057:Tomography, X-Ray Computed
Hepatotoxische Enzephalopathie versus Leigh-Syndrom--eine kasuistische Betrachtung.