Posterior tilting of the tibial component decreases femoral rollback in posterior-substituting knee replacement: a computer simulation study.
Posterior tilting of the tibial component is thought to increase the range of motion in posterior cruciate-retaining total knee replacement, but its effect on implant motion in posterior cruciate-substituting total knee replacement is unknown. This issue has become of interest recently because manufacturers have introduced instrumentation that produces a posteriorly tilted tibial cut for both implant types. The purpose of this study was to investigate how motion of posterior cruciate-substituting total knee replacement is affected when the tibial component is installed with posterior tilt. Sagittal plane implant motions were predicted from prosthesis geometry with use of a computer simulation in which the femoral condyles were assumed to sit in the bottoms of the tibial condylar wells when the knee was in extension. Rollback of the femoral component was produced by a cam-spine mechanism at higher angles of flexion. The simulations revealed that even small degrees of posterior tilt reduced rollback by limiting the interaction between the cam and spine. Tilting the component posteriorly by 5 degrees caused the cam to contact the spine at a knee flexion angle that was 18 degrees higher than with the untilted component. The results suggest that posterior tilting of the tibial component in posterior cruciate-substituting knee replacement may not produce the same beneficial effects that have been reported for the tilting of tibial components in posterior cruciate-retaining knee replacement.
Piazza
S J
SJ
Department of Mechanical Engineering, Northwestern University and Sensory Motor Performance Program, Rehabilitation Institute of Chicago, Illinois 60611, USA.
Delp
S L
SL
Stulberg
S D
SD
Stern
S H
SH
D019645:Arthroplasty, Replacement, Knee / Q000379:methods*; D003198:Computer Simulation; D005269:Femur / Q000502:physiology*; D006801:Humans; D007719:Knee Joint / Q000502:physiology; D008954:Models, Biological; D013977:Tibia / Q000502:physiology*