Prenatal cocaine exposure produces long-term impairments in brain serotonin function in rat offspring.
Battaglia
G
G
Department of Pharmacology, Stritch School of Medicine, Loyola University Chicago, Maywood, Illinois 60153, USA. gbattag@luc.edu
Cabrera-Vera
T M
TM
Van de Kar
L D
LD
Garcia
F
F
Vicentic
A
A
Pinto
W
W
D017371:8-Hydroxy-2-(di-n-propylamino)tetralin / Q000494:pharmacology; D000324:Adrenocorticotropic Hormone / Q000097:blood; D000818:Animals; D001921:Brain / Q000187:drug effects* / Q000502:physiology; D003042:Cocaine / Q000633:toxicity*; D005260:Female; D005865:Gestational Age; D008297:Male; D011247:Pregnancy; D011297:Prenatal Exposure Delayed Effects; D051381:Rats; D017207:Rats, Sprague-Dawley; D011985:Receptors, Serotonin / Q000502:physiology; D044263:Receptors, Serotonin, 5-HT1; D012083:Renin / Q000097:blood; D012701:Serotonin / Q000378:metabolism*; D010133:p-Chloroamphetamine / Q000494:pharmacology
D011985:Receptors, Serotonin; D044263:Receptors, Serotonin, 5-HT1; D012701:Serotonin; D010133:p-Chloroamphetamine; D017371:8-Hydroxy-2-(di-n-propylamino)tetralin; D000324:Adrenocorticotropic Hormone; D012083:Renin; D003042:Cocaine