Seronegative myasthenia plasmas and non-IgG fractions transiently inhibit nAChR function.
Plested
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Institute of Molecular Medicine, John Radcliffe Hospital, University of Oxford, Headington, United Kingdom.
Newsom-Davis
J
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Vincent
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D015740:Calcitonin Gene-Related Peptide / Q000494:pharmacology; D002217:Carbachol / Q000494:pharmacology*; D002460:Cell Line; D017868:Cyclic AMP-Dependent Protein Kinases / Q000037:antagonists & inhibitors; D004791:Enzyme Inhibitors / Q000494:pharmacology; D006801:Humans; D007074:Immunoglobulin G; D007546:Isoquinolines / Q000494:pharmacology; D009157:Myasthenia Gravis / Q000097:blood* / Q000276:immunology; D011978:Receptors, Nicotinic / Q000276:immunology / Q000502:physiology*; D011994:Recombinant Proteins / Q000187:drug effects / Q000378:metabolism; D012964:Sodium / Q000378:metabolism; D013449:Sulfonamides; D014162:Transfection
D004791:Enzyme Inhibitors; D007074:Immunoglobulin G; D007546:Isoquinolines; D011978:Receptors, Nicotinic; D011994:Recombinant Proteins; D013449:Sulfonamides; D002217:Carbachol; D012964:Sodium; D017868:Cyclic AMP-Dependent Protein Kinases; D015740:Calcitonin Gene-Related Peptide; C063509:N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide