Nitric oxide as a mediator of hemodynamic abnormalities and sodium and water retention in cirrhosis.
Martin
P Y
PY
Department of Medicine, University of Colorado Health Sciences Center, Denver 80262, USA.
Ginès
P
P
Schrier
R W
RW
D000818:Animals; D001201:Ascites / Q000209:etiology / Q000503:physiopathology; D005110:Extracellular Space / Q000378:metabolism; D006439:Hemodynamics; D006706:Homeostasis; D006801:Humans; D006975:Hypertension, Portal / Q000503:physiopathology; D007674:Kidney Diseases / Q000503:physiopathology; D008103:Liver Cirrhosis / Q000503:physiopathology*; D008106:Liver Cirrhosis, Experimental / Q000503:physiopathology*; D009569:Nitric Oxide / Q000037:antagonists & inhibitors / Q000502:physiology*; D019001:Nitric Oxide Synthase / Q000037:antagonists & inhibitors / Q000378:metabolism; D012964:Sodium / Q000378:metabolism*; D013152:Splanchnic Circulation / Q000502:physiology
D009569:Nitric Oxide; D012964:Sodium; D019001:Nitric Oxide Synthase