title: Hypothesis that tiagabine-induced NCSE is associated with GABAergic hyperfunction, with GABA(B) receptors playing a critical role, is supported by a case of generalized NCSE induced by baclofen.
authors.forename: G E
authors.initials: GE
authors.lastname: Solomon
authors.forename: D
authors.initials: D
authors.lastname: Labar
mesh_terms: D000927:Anticonvulsants / Q000009:adverse effects*; D001418:Baclofen / Q000009:adverse effects*; D006801:Humans; D009557:Nipecotic Acids / Q000009:adverse effects*; D018079:Receptors, GABA / Q000187:drug effects / Q000502:physiology; D013226:Status Epilepticus / Q000139:chemically induced*; D000078308:Tiagabine
chemical_list: D000927:Anticonvulsants; D009557:Nipecotic Acids; D018079:Receptors, GABA; D001418:Baclofen; D000078308:Tiagabine