title: Tooth wear in children: an investigation of etiological factors in children with cerebral palsy and gastroesophageal reflux.
abstract: This investigation was undertaken to establish the prevalence and distribution of tooth wear in different groups of medically compromised children and to assess the possible influence of gastroesophageal reflux, dietary factors, and parafunctional activity. Four groups of children including twenty-one with cerebral palsy were clinically examined and had in-depth, structured interviews, including information on medical history, medication, gastroesophageal reflux, feeding and drinking habits, parafunctional activity, and tooth-brushing procedures. All the children with cerebral palsy also had twenty-four-hour gastroesophageal pH monitoring. Of the fifty-one children in total, twenty-five had moderate or severe levels of dental erosion. No statistically significant differences were found between the groups as far as dietary influences, feeding habits, and tooth-brushing procedures. There was a significant association, however, between gastroesophageal reflux and erosion. It is concluded that in children with cerebral palsy this reflux may be much more important in the etiology of tooth surface loss than the parafunctional activity, which has classically been regarded as the cause.
authors.affiliation: Department of Paediatric Dentistry, University of Birmingham Dental School, United Kingdom.
authors.forename: L
authors.initials: L
authors.lastname: Shaw
authors.forename: S
authors.initials: S
authors.lastname: Weatherill
authors.forename: A
authors.initials: A
authors.lastname: Smith
mesh_terms: D001628:Beverages / Q000009:adverse effects; D002012:Bruxism / Q000150:complications; D002547:Cerebral Palsy / Q000150:complications*; D002648:Child; D017742:Dental Care for Chronically Ill; D005745:Gastric Acidity Determination; D005764:Gastroesophageal Reflux / Q000150:complications* / Q000209:etiology; D006801:Humans; D019217:Tooth Attrition / Q000209:etiology; D014077:Tooth Erosion / Q000209:etiology*