Absence of "original antigenic sin" in autoimmunity provides an unforeseen platform for immune therapy.
Steinman
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Department of Neurology and Neurological Sciences and Pediatrics, Becknman Center for Molecular and Genetic Medicine, Stanford University, Stanford, California 94305, USA. steinman@stanford.edu
D000818:Animals; D000940:Antigenic Variation; D000956:Antigens, Viral / Q000276:immunology; D001324:Autoantigens / Q000737:chemistry / Q000276:immunology*; D001327:Autoimmune Diseases / Q000276:immunology*; D015551:Autoimmunity / Q000276:immunology*; D002908:Chronic Disease; D004195:Disease Models, Animal; D018450:Disease Progression; D004681:Encephalomyelitis, Autoimmune, Experimental / Q000276:immunology*; D000939:Epitopes / Q000276:immunology*; D006801:Humans; D007111:Immunity, Cellular; D007114:Immunization; D016131:Lymphocyte Subsets / Q000276:immunology; D051379:Mice; D018448:Models, Immunological; D009103:Multiple Sclerosis / Q000276:immunology; D004676:Myelin Basic Protein / Q000276:immunology; D009185:Myelin Proteins / Q000737:chemistry / Q000276:immunology*; D018991:Myelin Proteolipid Protein / Q000276:immunology; D018992:Myelin-Associated Glycoprotein / Q000276:immunology; D063308:Myelin-Oligodendrocyte Glycoprotein; D017634:Self Tolerance / Q000276:immunology
D000956:Antigens, Viral; D001324:Autoantigens; D000939:Epitopes; C571326:MOG protein, human; C571327:Mog protein, mouse; D004676:Myelin Basic Protein; D009185:Myelin Proteins; D018991:Myelin Proteolipid Protein; D018992:Myelin-Associated Glycoprotein; D063308:Myelin-Oligodendrocyte Glycoprotein