Phosphorylation substrates and altered signalling in leukemias caused by BCR/ABL.
Groffen
J
J
Department of Pathology, Childrens Hospital of Los Angeles, CA, USA.
de Jong
R
R
Haataja
L
L
Kaartinen
V
V
Heisterkamp
N
N
D048868:Adaptor Proteins, Signal Transducing; D000818:Animals; D001798:Blood Proteins / Q000737:chemistry; D016044:Fusion Proteins, bcr-abl / Q000737:chemistry / Q000235:genetics / Q000502:physiology*; D005838:Genotype; D006801:Humans; D015464:Leukemia, Myelogenous, Chronic, BCR-ABL Positive / Q000235:genetics / Q000378:metabolism*; D051379:Mice; D018345:Mice, Knockout; D009363:Neoplasm Proteins / Q000378:metabolism*; D009687:Nuclear Proteins / Q000378:metabolism; D010750:Phosphoproteins / Q000378:metabolism*; D010766:Phosphorylation; D011499:Protein Processing, Post-Translational / Q000235:genetics / Q000502:physiology*; D015398:Signal Transduction / Q000235:genetics / Q000502:physiology*; D018909:src Homology Domains
D048868:Adaptor Proteins, Signal Transducing; D001798:Blood Proteins; C082869:CRKL protein; C099942:NEDD9 protein, human; D009363:Neoplasm Proteins; D009687:Nuclear Proteins; D010750:Phosphoproteins; C039077:platelet protein P47; D016044:Fusion Proteins, bcr-abl