A Fas pathway to pulmonary fibrosis.
Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts 02115, USA. hchapman@rics.bwh.harvard.edu
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Chapman
D000818:Animals; D017209:Apoptosis; D001761:Bleomycin / Q000633:toxicity; D016207:Cytokines / Q000502:physiology; D053222:Fas Ligand Protein; D006801:Humans; D017563:Lung Diseases, Interstitial / Q000276:immunology / Q000473:pathology; D008562:Membrane Glycoproteins / Q000502:physiology; D051379:Mice; D019463:Mice, Inbred MRL lpr; D008817:Mice, Mutant Strains; D008954:Models, Biological; D011650:Pulmonary Alveoli / Q000473:pathology*; D011658:Pulmonary Fibrosis / Q000139:chemically induced / Q000209:etiology* / Q000276:immunology / Q000473:pathology; D019014:fas Receptor / Q000276:immunology / Q000502:physiology*
D016207:Cytokines; C505661:FASLG protein, human; D053222:Fas Ligand Protein; C505658:Fasl protein, mouse; D008562:Membrane Glycoproteins; D019014:fas Receptor; D001761:Bleomycin