ATM and lymphoid malignancies; use of oriented peptide libraries to identify novel substrates of ATM critical in downstream signaling pathways.
Center for Blood Research, Harvard Medical School, USA.
G A
GA
Rathbun
Y
Y
Ziv
J H
JH
Lai
D
D
Hill
R H
RH
Abraham
Y
Y
Shiloh
L C
LC
Cantley
D001260:Ataxia Telangiectasia / Q000150:complications / Q000235:genetics; D064007:Ataxia Telangiectasia Mutated Proteins; D018797:Cell Cycle Proteins; D002471:Cell Transformation, Neoplastic; D004268:DNA-Binding Proteins; D006801:Humans; D008223:Lymphoma / Q000209:etiology / Q000235:genetics*; D009154:Mutation; D019151:Peptide Library; D017346:Protein Serine-Threonine Kinases; D011506:Proteins / Q000235:genetics* / Q000378:metabolism; D015398:Signal Transduction; D025521:Tumor Suppressor Proteins
D018797:Cell Cycle Proteins; D004268:DNA-Binding Proteins; D019151:Peptide Library; D011506:Proteins; D025521:Tumor Suppressor Proteins; C576324:ATM protein, human; D064007:Ataxia Telangiectasia Mutated Proteins; D017346:Protein Serine-Threonine Kinases