Mechanisms of remodeling of gap junction distributions and the development of anatomic substrates of arrhythmias.
Saffitz
J E
JE
Department of Pathology, Washington University School of Medicine, St. Louis, MO 63110, USA. saffitz@pathology.wustl.edu
Schuessler
R B
RB
Yamada
K A
KA
D000818:Animals; D001145:Arrhythmias, Cardiac / Q000378:metabolism* / Q000473:pathology; D006332:Cardiomegaly / Q000378:metabolism / Q000473:pathology; D002908:Chronic Disease; D018031:Connexin 43 / Q000235:genetics / Q000378:metabolism; D017629:Gap Junctions / Q000378:metabolism*; D005786:Gene Expression Regulation; D006168:Guinea Pigs; D006352:Heart Ventricles; D006801:Humans; D007473:Ion Channels / Q000235:genetics*; D017136:Ion Transport; D009206:Myocardium / Q000378:metabolism* / Q000473:pathology; D051381:Rats; D020257:Ventricular Remodeling / Q000235:genetics*
D018031:Connexin 43; D007473:Ion Channels