[Electrophile counter-attack response].
Ishii
T
T
Department of Biochemistry, University of Tsukuba, Japan. teishii@md.tsukuba.ac.jp
Itoh
K
K
D000818:Animals; D000975:Antioxidants / Q000494:pharmacology; D002083:Butylated Hydroxyanisole / Q000494:pharmacology; D002273:Carcinogens / Q000633:toxicity*; D003553:Cystine / Q000378:metabolism; D004742:Enhancer Elements, Genetic; D004790:Enzyme Induction / Q000187:drug effects; D005978:Glutathione / Q000378:metabolism; D006419:Heme Oxygenase (Decyclizing) / Q000096:biosynthesis / Q000235:genetics*; D051547:Heme Oxygenase-1; D006801:Humans; D017737:Macrophages, Peritoneal / Q000378:metabolism; D008565:Membrane Proteins; D018384:Oxidative Stress; D010544:Peroxidases / Q000096:biosynthesis / Q000235:genetics*; D054464:Peroxiredoxins; D014157:Transcription Factors / Q000502:physiology*
D000975:Antioxidants; D002273:Carcinogens; D008565:Membrane Proteins; D014157:Transcription Factors; D002083:Butylated Hydroxyanisole; D003553:Cystine; D010544:Peroxidases; D054464:Peroxiredoxins; C496541:HMOX1 protein, human; D006419:Heme Oxygenase (Decyclizing); D051547:Heme Oxygenase-1; D005978:Glutathione