How does knocking out angiotensin II activity reduce renal injury in mice?
Lafayette
R A
RA
Stanford University School of Medicine, Stanford, CA, USA.
D000804:Angiotensin II / Q000502:physiology*; D000818:Animals; D001485:Basement Membrane / Q000276:immunology; D006977:Hypertension, Renal / Q000276:immunology; D018836:Inflammation Mediators / Q000097:blood; D007676:Kidney Failure, Chronic / Q000276:immunology*; D007678:Kidney Glomerulus / Q000276:immunology; D051379:Mice; D008815:Mice, Inbred Strains; D044140:Receptor, Angiotensin, Type 1; D011945:Receptors, Angiotensin / Q000172:deficiency / Q000502:physiology
D018836:Inflammation Mediators; D044140:Receptor, Angiotensin, Type 1; D011945:Receptors, Angiotensin; D000804:Angiotensin II