[Lipid peroxidation and function of some antioxidant enzyme systems in corn and oats in acute injury by hydrogen fluoride].
Experimental research showed the HF main harmful effect at the maximal exposure time (12 h). Intensity of chemiluminescence processes was enhanced almost in 1.5-fold. In the maize leaves the reduced chemiluminescence intensity was observed under the maximal toxicant concentration (1 mg/m3) effect, whereas the further growth of chemiluminescence intensity was observed in the oat plants. The total amount of the lipid peroxidation products was increased dependently on the toxicant concentration in all experimental variants. Changes in the studied enzymatic system activities were multidirected. Analysis of all activities dynamics for the SOD, GGTP, GST, GR and GP showed that GGTP and sometimes GP were involved more rapidly then SOD into the processes of detoxication of lipid peroxidation products and metabolites formed under the HF effect. However, the correlation between changes both in the SOD and GP activities failed to be found. The obtained data give a basis to conclude that during period between 8 and 12 h of HF exposure the essential changes in the activities of SOD and glutathione enzymatic systems took place as an adaptive response of plant cell to the investigated stress-factor impact.
Grishko
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VN
Syshcheikov
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DV
D000975:Antioxidants / Q000378:metabolism; D018554:Avena / Q000187:drug effects* / Q000201:enzymology / Q000378:metabolism; D005980:Glutathione Reductase / Q000378:metabolism; D005982:Glutathione Transferase / Q000378:metabolism; D006858:Hydrofluoric Acid / Q000633:toxicity*; D015227:Lipid Peroxidation / Q000187:drug effects*; D013482:Superoxide Dismutase / Q000378:metabolism; D003313:Zea mays / Q000187:drug effects* / Q000201:enzymology / Q000378:metabolism; D005723:gamma-Glutamyltransferase / Q000378:metabolism
D000975:Antioxidants; D013482:Superoxide Dismutase; D005980:Glutathione Reductase; D005723:gamma-Glutamyltransferase; D005982:Glutathione Transferase; D006858:Hydrofluoric Acid