Temporary axonal conduction block and axonal loss in inflammatory neurological disease. A potential role for nitric oxide?
Kapoor
R
R
Department of Clinical Neurosciences, Guy's, King's & St. Thomas' School of Medicine, King's College, London, United Kingdom.
Davies
M
M
Smith
K J
KJ
D000200:Action Potentials; D000818:Animals; D001369:Axons / Q000187:drug effects / Q000473:pathology / Q000502:physiology*; D004558:Electric Stimulation; D005260:Female; D005727:Ganglia, Spinal / Q000502:physiology; D007249:Inflammation; D008297:Male; D009422:Nervous System Diseases / Q000473:pathology / Q000503:physiopathology*; D009569:Nitric Oxide / Q000502:physiology*; D020030:Nitric Oxide Donors / Q000494:pharmacology*; D009589:Nitrogen Oxides; D009603:Nitroso Compounds / Q000494:pharmacology; D051381:Rats; D011917:Rats, Inbred Lew; D017207:Rats, Sprague-Dawley; D017208:Rats, Wistar; D013096:Spermine / Q000031:analogs & derivatives / Q000494:pharmacology; D013116:Spinal Cord / Q000187:drug effects / Q000502:physiology; D013126:Spinal Nerve Roots / Q000187:drug effects / Q000502:physiology
D020030:Nitric Oxide Donors; D009589:Nitrogen Oxides; D009603:Nitroso Compounds; C091861:spermine nitric oxide complex; C094210:2,2'-(hydroxynitrosohydrazono)bis-ethanamine; D013096:Spermine; D009569:Nitric Oxide