Aspirin-triggered 15-epi-lipoxin A4 and novel lipoxin B4 stable analogs inhibit neutrophil-mediated changes in vascular permeability.
Serhan
C N
CN
Department of Anesthesia, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts 02115, USA.
Takano
T
T
Clish
C B
CB
Gronert
K
K
Petasis
N
N
D000818:Animals; D001241:Aspirin / Q000494:pharmacology*; D002199:Capillary Permeability / Q000187:drug effects* / Q000502:physiology*; D002465:Cell Movement / Q000187:drug effects; D005233:Fatty Alcohols / Q000494:pharmacology; D006018:Glycols / Q000494:pharmacology; D006893:Hydroxyeicosatetraenoic Acids / Q000737:chemistry / Q000502:physiology*; D044045:Lipoxins; D051379:Mice; D008807:Mice, Inbred BALB C; D009504:Neutrophils / Q000187:drug effects* / Q000502:physiology*; D018102:Receptors, Leukotriene B4 / Q000037:antagonists & inhibitors / Q000502:physiology
D005233:Fatty Alcohols; D006018:Glycols; D006893:Hydroxyeicosatetraenoic Acids; D044045:Lipoxins; D018102:Receptors, Leukotriene B4; C040527:lipoxin A4; C062137:U 75302; C042651:lipoxin B4; D001241:Aspirin