beta-adrenergic receptor signaling: an acute compensatory adjustment-inappropriate for the chronic stress of heart failure? Insights from Gsalpha overexpression and other genetically engineered animal models.
Vatner
S F
SF
Sigfried and Janet Weis Center for Research, The Pennsylvania State University College of Medicine, Danville, PA 17822-2601, USA.
Vatner
D E
DE
Homcy
C J
CJ
D000818:Animals; D002908:Chronic Disease; D004195:Disease Models, Animal; D019205:GTP-Binding Protein alpha Subunits, Gs / Q000235:genetics*; D015870:Gene Expression; D005818:Genetic Engineering; D006333:Heart Failure / Q000235:genetics / Q000503:physiopathology*; D011943:Receptors, Adrenergic, beta / Q000502:physiology*; D015398:Signal Transduction / Q000502:physiology*; D013312:Stress, Physiological / Q000235:genetics / Q000503:physiopathology*
D011943:Receptors, Adrenergic, beta; D019205:GTP-Binding Protein alpha Subunits, Gs