Wnts as retrograde signals for axon and growth cone differentiation.
Burden
S J
SJ
Molecular Neurobiology Program, Skirball Institute of Biomolecular Medicine, New York University Medical School, New York, New York 10016, USA. burden@saturn.med.nyu.edu
D018171:Agrin / Q000502:physiology; D001369:Axons / Q000502:physiology*; D017871:Calcium-Calmodulin-Dependent Protein Kinases / Q000037:antagonists & inhibitors; D002454:Cell Differentiation; D002525:Cerebellar Cortex / Q000166:cytology* / Q000196:embryology; D038362:Glycogen Synthase Kinase 3; D020439:Growth Cones / Q000502:physiology*; D009412:Nerve Fibers / Q000502:physiology*; D009419:Nerve Tissue Proteins / Q000502:physiology*; D009469:Neuromuscular Junction / Q000648:ultrastructure; D009473:Neuronal Plasticity; D010766:Phosphorylation; D011499:Protein Processing, Post-Translational; D011518:Proto-Oncogene Proteins / Q000502:physiology*; D015398:Signal Transduction; D013569:Synapses / Q000502:physiology; D051153:Wnt Proteins; D029961:Zebrafish Proteins
D018171:Agrin; D009419:Nerve Tissue Proteins; D011518:Proto-Oncogene Proteins; D051153:Wnt Proteins; D029961:Zebrafish Proteins; D017871:Calcium-Calmodulin-Dependent Protein Kinases; D038362:Glycogen Synthase Kinase 3