title: Fetal origins of reduced arterial distensibility in the donor twin in twin-twin transfusion syndrome.
abstract: Twin-twin transfusion syndrome permits investigation of vascular programming independent of genetic influence. Arterial distensibiity was lower in the donor twin during infancy, implying the intrauterine vascular remodelling might result in raised cardiac afterload and could influence later cardiovascular health.
authors.forename: Y F
authors.initials: YF
authors.lastname: Cheung
authors.forename: M J
authors.initials: MJ
authors.lastname: Taylor
authors.forename: N M
authors.initials: NM
authors.lastname: Fisk
authors.forename: A N
authors.initials: AN
authors.lastname: Redington
authors.forename: H M
authors.initials: HM
authors.lastname: Gardiner
mesh_terms: D001783:Blood Flow Velocity; D001794:Blood Pressure; D001916:Brachial Artery / Q000503:physiopathology*; D004200:Diseases in Twins; D005260:Female; D005330:Fetofetal Transfusion / Q000503:physiopathology*; D006801:Humans; D007223:Infant; D007231:Infant, Newborn; D017156:Photoplethysmography; D011247:Pregnancy; D011673:Pulsatile Flow / Q000502:physiology; D017534:Radial Artery / Q000503:physiopathology*; D014429:Twins, Dizygotic; D014430:Twins, Monozygotic; D014655:Vascular Resistance / Q000502:physiology