title: [Nitrogen monoxide in the mechanisms of cardiovascular functional resistance to emotional stress].
abstract: The paper presents the results of examination of the mechanisms of development of different physiological dysfunctions (diminished cardiac electrical stability, fibrillation threshold fall) under emotional stress. Search was made for their drug correction (nitric oxide precursor L-arginine, the NO synthase blocker Ng-methyl-L-arginine and sodium nitrate). Experiments have indicated that L-arginine and sodium nitrate restore cardiac electrical stability. The normalizing effects of NO donors depended on the stage of cardiac disorders.
authors.forename: S I
authors.initials: SI
authors.lastname: Kashtanov
authors.forename: M A
authors.initials: MA
authors.lastname: Zviagintseva
authors.forename: I L
authors.initials: IL
authors.lastname: Kosharskaia
authors.forename: L V
authors.initials: LV
authors.lastname: Mezentseva
authors.forename: V N
authors.initials: VN
authors.lastname: Gurin
mesh_terms: D000208:Acute Disease; D000818:Animals; D002320:Cardiovascular Physiological Phenomena / Q000187:drug effects; D002908:Chronic Disease; D004195:Disease Models, Animal; D006439:Hemodynamics / Q000187:drug effects; D008297:Male; D009569:Nitric Oxide / Q000502:physiology*; D020030:Nitric Oxide Donors / Q000494:pharmacology; D019001:Nitric Oxide Synthase / Q000037:antagonists & inhibitors; D051381:Rats; D017208:Rats, Wistar; D013315:Stress, Psychological / Q000209:etiology* / Q000503:physiopathology
chemical_list: D020030:Nitric Oxide Donors; D009569:Nitric Oxide; D019001:Nitric Oxide Synthase