Restenosis after percutaneous coronary interventions and infection.
Infectious agents may directly or indirectly (through the response of the host's immune system) modulate the growth of vascular cells. Local and/or systemic increase of cytokines could influence the extent of (re-)stenosis in the vascular tree. Further studies in this field may identify patients at a high risk for atherogenesis and restenosis. Their results should be helpful in treating restenosis after percutaneous coronary interventions.
Herzum
M
M
Department of Internal Medicine, Philipps University of Marburg. herzum@mailer.uni-marburg.de
Schaefer
J R
JR
Simon
B
B
Menz
V
V
Noll
B
B
Maisch
B
B
D015906:Angioplasty, Balloon, Coronary; D000818:Animals; D001424:Bacterial Infections / Q000150:complications*; D003327:Coronary Disease / Q000209:etiology / Q000628:therapy*; D016207:Cytokines / Q000502:physiology; D003586:Cytomegalovirus Infections / Q000150:complications; D006566:Herpesviridae Infections / Q000150:complications; D006801:Humans; D015994:Incidence; D024510:Muscle Development; D009131:Muscle, Smooth, Vascular / Q000166:cytology / Q000254:growth & development; D012008:Recurrence; D012307:Risk Factors; D014777:Virus Diseases / Q000150:complications*
D016207:Cytokines