Non-MHC-linked genetic polymorphism controls selective development of Th2 but not inhibition of Th1 cells by soluble antigen administration.
Galbiati
F
F
Roche Milano Ricerche, Milan, Italy.
Adorini
L
L
D000818:Animals; D000941:Antigens / Q000008:administration & dosage / Q000276:immunology*; D004198:Disease Susceptibility / Q000276:immunology; D006801:Humans; D015847:Interleukin-4 / Q000276:immunology; D018320:Leishmania major / Q000276:immunology; D016773:Leishmaniasis, Cutaneous / Q000276:immunology; D008285:Major Histocompatibility Complex / Q000276:immunology*; D011110:Polymorphism, Genetic; D018123:Receptors, Interleukin / Q000276:immunology; D053707:Receptors, Interleukin-12; D018417:Th1 Cells / Q000276:immunology*; D018418:Th2 Cells / Q000276:immunology*
D000941:Antigens; D018123:Receptors, Interleukin; D053707:Receptors, Interleukin-12; D015847:Interleukin-4