title: Restenosis after percutaneous coronary interventions and infection.
abstract: Infectious agents may directly or indirectly (through the response of the host's immune system) modulate the growth of vascular cells. Local and/or systemic increase of cytokines could influence the extent of (re-)stenosis in the vascular tree. Further studies in this field may identify patients at a high risk for atherogenesis and restenosis. Their results should be helpful in treating restenosis after percutaneous coronary interventions.
authors.affiliation: Department of Internal Medicine, Philipps University of Marburg. herzum@mailer.uni-marburg.de
authors.forename: M
authors.initials: M
authors.lastname: Herzum
authors.forename: J R
authors.initials: JR
authors.lastname: Schaefer
authors.forename: B
authors.initials: B
authors.lastname: Simon
authors.forename: V
authors.initials: V
authors.lastname: Menz
authors.forename: B
authors.initials: B
authors.lastname: Noll
authors.forename: B
authors.initials: B
authors.lastname: Maisch
mesh_terms: D015906:Angioplasty, Balloon, Coronary; D000818:Animals; D001424:Bacterial Infections / Q000150:complications*; D003327:Coronary Disease / Q000209:etiology / Q000628:therapy*; D016207:Cytokines / Q000502:physiology; D003586:Cytomegalovirus Infections / Q000150:complications; D006566:Herpesviridae Infections / Q000150:complications; D006801:Humans; D015994:Incidence; D024510:Muscle Development; D009131:Muscle, Smooth, Vascular / Q000166:cytology / Q000254:growth & development; D012008:Recurrence; D012307:Risk Factors; D014777:Virus Diseases / Q000150:complications*
chemical_list: D016207:Cytokines