A novel proteolytic mechanism for termination of the CA2+ signalling evoked by proteinase-activated receptor-1 (PAR-1) in rat astrocytes.
Ubl
J J
JJ
Otto-von-Guericke Universität Magdeburg, Medizinische Fakultät, Germany.
Reiser
G
G
D000818:Animals; D001253:Astrocytes / Q000187:drug effects* / Q000378:metabolism; D020013:Calcium Signaling / Q000187:drug effects / Q000502:physiology*; D008024:Ligands; D008954:Models, Biological; D009842:Oligopeptides / Q000494:pharmacology*; D017434:Protein Structure, Tertiary; D051381:Rats; D044463:Receptor, PAR-1; D018179:Receptors, Thrombin / Q000819:agonists / Q000502:physiology*; D013820:Thermolysin / Q000494:pharmacology; D013917:Thrombin / Q000494:pharmacology; D014360:Trypsin Inhibitor, Kunitz Soybean / Q000494:pharmacology
C415750:Ala-4-fluoro-Phe-Arg-cyclohexyl-Ala-homoArg-Tyr-NH2; D008024:Ligands; D009842:Oligopeptides; D044463:Receptor, PAR-1; D018179:Receptors, Thrombin; D014360:Trypsin Inhibitor, Kunitz Soybean; D013917:Thrombin; D013820:Thermolysin