Primary aldosteronism in a patient with familial adenomatous polyposis.
Patients with familial adenomatous polyposis (FAP) frequently have extracolonic manifestations of their disease. Prior reports have indicated an increased prevalence of adrenal lesions in patients with FAP. Although most of the adrenal lesions represent nonfunctioning adenomas, some patients have had hypercortisolism due to adrenocortical carcinoma or bilateral nodular hyperplasia. We present a case of a patient with FAP who had mineralocorticoid excess due to an aldosterone-producing adrenocortical adenoma.
Alexander
G L
GL
Division of Gastroenterology and Hepatology and Internal Medicine, Mayo Clinic, Rochester, Minn 55905, USA.
Thompson
G B
GB
Schwartz
D A
DA
D011125:Adenomatous Polyposis Coli / Q000473:pathology* / Q000601:surgery; D000306:Adrenal Cortex Neoplasms / Q000150:complications* / Q000378:metabolism / Q000556:secondary* / Q000601:surgery; D000315:Adrenalectomy; D000450:Aldosterone / Q000097:blood / Q000378:metabolism* / Q000652:urine; D006801:Humans; D006929:Hyperaldosteronism / Q000209:etiology* / Q000378:metabolism; D008297:Male; D008875:Middle Aged; D016896:Treatment Outcome
D000450:Aldosterone