[An increased proinflammatory activity is inherent in insulin resistance].
Fernández-Real
J M
JM
Unidad de Diabetes, Endocrinología y Nutrición (UDEN), Hospital Universitario de Girona Dr. Josep Trueta. endocrino@htrueta.scs.es
Ricart
W
W
D017667:Adipocytes / Q000502:physiology; D000273:Adipose Tissue / Q000166:cytology / Q000503:physiopathology; D000328:Adult; D000818:Animals; D050260:Carbohydrate Metabolism; D002478:Cells, Cultured; D003921:Diabetes Mellitus, Experimental / Q000378:metabolism / Q000503:physiopathology; D003924:Diabetes Mellitus, Type 2 / Q000235:genetics / Q000378:metabolism / Q000503:physiopathology*; D005260:Female; D015870:Gene Expression; D005838:Genotype; D006801:Humans; D007249:Inflammation / Q000209:etiology; D018836:Inflammation Mediators / Q000502:physiology*; D007328:Insulin / Q000502:physiology*; D007333:Insulin Resistance / Q000235:genetics / Q000502:physiology; D015850:Interleukin-6 / Q000235:genetics / Q000502:physiology; D007515:Islets of Langerhans / Q000503:physiopathology; D050356:Lipid Metabolism; D008099:Liver / Q000503:physiopathology; D008297:Male; D051379:Mice; D018345:Mice, Knockout; D008820:Mice, Obese; D009154:Mutation; D009765:Obesity / Q000235:genetics / Q000378:metabolism / Q000503:physiopathology*; D011110:Polymorphism, Genetic; D014409:Tumor Necrosis Factor-alpha / Q000235:genetics / Q000502:physiology
D018836:Inflammation Mediators; D007328:Insulin; D015850:Interleukin-6; D014409:Tumor Necrosis Factor-alpha