The trans-spliced L30 ribosomal protein mRNA of Trypanosoma brucei is not subject to autogenous feedback control at the messenger RNA level.
Wilson
K
K
Fairchild D305, Department of Microbiology and Immunology, School of Medicine, Stanford University, Stanford, CA 94305-5402, USA.
Uyetake
L
L
Boothroyd
J C
JC
D000818:Animals; D001483:Base Sequence; D005246:Feedback; D005786:Gene Expression Regulation; D017125:Genes, Protozoan; D005822:Genetic Vectors; D051379:Mice; D008969:Molecular Sequence Data; D016366:Open Reading Frames; D014176:Protein Biosynthesis; D012326:RNA Splicing; D012333:RNA, Messenger / Q000235:genetics* / Q000378:metabolism; D016053:RNA, Protozoan / Q000235:genetics* / Q000378:metabolism; D012269:Ribosomal Proteins / Q000235:genetics* / Q000378:metabolism; D014170:Transformation, Genetic; D014346:Trypanosoma brucei brucei / Q000235:genetics* / Q000378:metabolism
D012333:RNA, Messenger; D016053:RNA, Protozoan; D012269:Ribosomal Proteins; C035201:ribosomal protein L30