title: [Biliary acute pancreatitis: an etiopathology and evolution review of 148 cases].
abstract: BACKGROUND
  A retrospective analysis of the etiologic factors and physiopathogenic mechanisms underlying an acute biliary pancreatitis episode put in evidence the complexity of the attempt to acquire a clear understanding of the entity. In this presentation the authors try to re-examine the main factors involved in the triggering of the disease. Besides the classic theories and their own approach to the management of an acute pancreatic inflammation episode are discussed.
  
  
  OBJECTIVE
  The main purpose of this endeavor was to identify and discuss the etiopathogenic mechanisms that were prevalent in a series of 148 patients observed and treated in a 10 years period. Besides, another distinctive aim was to analyze their evolution and somehow to try to assess their probable prognosis. PLACE OF APPLICATION: Close community.
  
  
  METHODS
  The whole group of patients that were admitted with the diagnosis of acute pancreatitis in the time period comprised between 1987-1997.
  
  
  METHODS
  The acute pancreatitis subgroups and their respective number of patient included were the following: BILIARY ACUTE PANCREATITIS 140 CASES POST ERCP 8 CASES CONCLUSIONS: As a result of the present reviewing endeavour several observations deserve to be pointed out: a. The pancreatic gland undoubtedly is a neuroendocrine organ, that is subjected to complex neural and hormonal influence. b. Undeniably, the involvement of the autonomic nervous system in the physiopathogenic mechanism of acute pancreatitis has been surprisingly disregarded. c. The biliary acute pancreatitis variant is the most frequent. What we consider a simplification is to accept the Opie's postulation without taking into account the intermediate steps, centered on autonomic reflexes, that ultimately lead to the acute inflammatory lesions. d. Although without an absolute proof, it is undeniable that "stress" is a primary etiology in some cases of acute pancreatitis. e. We favor the idea that the pancreas functional status influences on the extension and intensity degree of the acute pancreatic inflammatory lesions.
authors.affiliation: Servicio de Cirugía General, Hospital Churruca.
authors.forename: H A
authors.initials: HA
authors.lastname: García
authors.forename: C
authors.initials: C
authors.lastname: Monti
authors.forename: S
authors.initials: S
authors.lastname: Del Giudice
authors.forename: G
authors.initials: G
authors.lastname: Eufemio
authors.forename: F
authors.initials: F
authors.lastname: Gasali
authors.forename: A
authors.initials: A
authors.lastname: Maschietto
authors.forename: N
authors.initials: N
authors.lastname: Molinelli Wells
authors.forename: M
authors.initials: M
authors.lastname: García
authors.forename: O M
authors.initials: OM
authors.lastname: Tiscornia
mesh_terms: D000208:Acute Disease; D000328:Adult; D000368:Aged; D000369:Aged, 80 and over; D001649:Bile Duct Diseases / Q000150:complications*; D002760:Cholangiopancreatography, Endoscopic Retrograde / Q000009:adverse effects*; D005260:Female; D006801:Humans; D008297:Male; D008875:Middle Aged; D010195:Pancreatitis / Q000209:etiology* / Q000503:physiopathology; D012189:Retrospective Studies