The motor circuit of the human basal ganglia reconsidered.
The standard model of human basal ganglia organization was introduced in the 1980s on the basis of animal experiments and clinical experience of various human motor disorders. This paper reviews evidence from various sources which suggests that this standard model only incompletely accounts for aspects of basal ganglia function, and thus requires modification.
Foley
P
P
Department of Psychiatry, University of Würzburg, Federal Republic of Germany.
Riederer
P
P
D015632:1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; D000818:Animals; D001479:Basal Ganglia / Q000502:physiology*; D015259:Dopamine Agents; D005917:Globus Pallidus / Q000503:physiopathology; D006801:Humans; D008959:Models, Neurological; D009043:Motor Activity / Q000502:physiology*; D009434:Neural Pathways / Q000502:physiology; D010300:Parkinson Disease / Q000503:physiopathology; D010302:Parkinson Disease, Secondary / Q000139:chemically induced; D009435:Synaptic Transmission / Q000502:physiology
D015259:Dopamine Agents; D015632:1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine