The role of spinal nitric oxide in the control of spontaneous pain following nociceptive input.
Hoheisel
U
U
Institut für Anatomie und Zellbiologie, Heidelberg, Germany.
Mense
S
S
D000200:Action Potentials / Q000187:drug effects / Q000502:physiology; D000818:Animals; D002452:Cell Count / Q000592:standards; D004558:Electric Stimulation / Q000009:adverse effects; D009220:Myositis / Q000139:chemically induced / Q000150:complications; D009252:NADPH Dehydrogenase / Q000378:metabolism; D009569:Nitric Oxide / Q000378:metabolism*; D019001:Nitric Oxide Synthase / Q000037:antagonists & inhibitors / Q000378:metabolism; D009619:Nociceptors / Q000166:cytology / Q000378:metabolism*; D010146:Pain / Q000378:metabolism* / Q000473:pathology / Q000503:physiopathology; D020671:Posterior Horn Cells / Q000473:pathology / Q000503:physiopathology; D051381:Rats; D017207:Rats, Sprague-Dawley; D012584:Sciatic Nerve / Q000378:metabolism / Q000473:pathology / Q000503:physiopathology
D009569:Nitric Oxide; D019001:Nitric Oxide Synthase; D009252:NADPH Dehydrogenase