The diuretic- and Bartter-like salt-losing tubulopathies.
Jeck
N
N
Department of Pediatrics, Philipps University, Marburg, Germany.
Konrad
M
M
Hess
M
M
Seyberth
H W
HW
D027321:Anion Transport Proteins; D001477:Bartter Syndrome / Q000378:metabolism*; D001581:Benzothiadiazines; D018118:Chloride Channels / Q000235:genetics; D004232:Diuretics / Q000009:adverse effects*; D005665:Furosemide / Q000009:adverse effects; D006579:Heterozygote; D006720:Homozygote; D006801:Humans; D007231:Infant, Newborn; D007674:Kidney Diseases / Q000139:chemically induced* / Q000378:metabolism*; D007684:Kidney Tubules; D008565:Membrane Proteins; D009154:Mutation / Q000502:physiology; D015221:Potassium Channels / Q000235:genetics; D024661:Potassium Channels, Inwardly Rectifying; D012965:Sodium Chloride / Q000378:metabolism*; D049993:Sodium Chloride Symporter Inhibitors / Q000009:adverse effects
D027321:Anion Transport Proteins; D001581:Benzothiadiazines; C412733:CLCNKB protein, human; D018118:Chloride Channels; D004232:Diuretics; C086989:KCNJ1 protein, human; D008565:Membrane Proteins; D015221:Potassium Channels; D024661:Potassium Channels, Inwardly Rectifying; D049993:Sodium Chloride Symporter Inhibitors; D012965:Sodium Chloride; D005665:Furosemide