Inhaled nitric oxide ameliorates postoperative acute graft dysfunction after living-donor lobar lung transplantation.
Inhaled nitric oxide, a selective pulmonary vasodilator, decreases mean pulmonary artery pressure and pulmonary vascular resistance and improves oxygenation in previously unobtainable ways without significant changes in systemic hemodynamics. We report successful treatment of a patient with acute graft dysfunction after living-donor lobar lung transplantation using nitric oxide inhalation.
Yoshikawa
T
T
Second Department of Surgery, Okayama University School of Medicine, 2-5-1 Shikatacho, Okayama 700-8558, Japan.
Date
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H
Yamashita
M
M
Nagahiro
I
I
Aoe
M
M
Shimizu
N
N
D000208:Acute Disease; D000280:Administration, Inhalation; D000328:Adult; D005260:Female; D006801:Humans; D006976:Hypertension, Pulmonary / Q000150:complications; D019520:Living Donors; D008171:Lung Diseases / Q000188:drug therapy* / Q000503:physiopathology; D016040:Lung Transplantation; D009569:Nitric Oxide / Q000008:administration & dosage*; D010101:Oxygen Consumption; D011184:Postoperative Period; D011654:Pulmonary Edema / Q000188:drug therapy; D014665:Vasodilator Agents / Q000008:administration & dosage*
D014665:Vasodilator Agents; D009569:Nitric Oxide