NF-kappaB: pivotal mediator or innocent bystander in atherogenesis?
Collins
T
T
Department of Pathology, Brigham and Women's Hospital and Harvard Medical School, 221 Longwood Avenue, Boston, Massachusetts 02115, USA. tcollins@rics.bwh.harvard.edu
Cybulsky
M I
MI
D000818:Animals; D001011:Aorta / Q000378:metabolism / Q000473:pathology; D017209:Apoptosis; D001161:Arteriosclerosis / Q000209:etiology* / Q000378:metabolism / Q000473:pathology; D048909:Diabetes Complications; D004730:Endothelium, Vascular / Q000378:metabolism / Q000382:microbiology; D006439:Hemodynamics; D006710:Homocysteine / Q000378:metabolism; D006801:Humans; D006973:Hypertension / Q000150:complications; D007962:Leukocytes / Q000502:physiology; D016328:NF-kappa B / Q000502:physiology*; D018384:Oxidative Stress; D012307:Risk Factors; D019010:Vascular Cell Adhesion Molecule-1 / Q000096:biosynthesis
D016328:NF-kappa B; D019010:Vascular Cell Adhesion Molecule-1; D006710:Homocysteine