SmcR-dependent regulation of adaptive phenotypes in Vibrio vulnificus.
Vibrio vulnificus contains homologues of the V. harveyi luxR and luxS genes. A null mutation in smcR (luxR) resulted in a defect in starvation survival, inhibition of starvation-induced maintenance of culturability that occurs when V. vulnificus is starved prior to low-temperature incubation, and increased expression of stationary-phase phenotypes.
McDougald
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School of Microbiology and Immunology, The University of New South Wales, Sydney, New South Wales, 2052, Australia.
Rice
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Kjelleberg
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D000220:Adaptation, Biological / Q000235:genetics*; D001426:Bacterial Proteins / Q000235:genetics; D013437:Carbon-Sulfur Lyases; D020689:Exopeptidases; D005798:Genes, Bacterial; D008163:Luminescent Measurements; D010641:Phenotype; D012097:Repressor Proteins / Q000235:genetics; D015398:Signal Transduction; D013217:Starvation; D015534:Trans-Activators / Q000235:genetics / Q000378:metabolism*; D014733:Vibrio / Q000235:genetics*
D001426:Bacterial Proteins; D012097:Repressor Proteins; D015534:Trans-Activators; C410093:smcR protein, Vibrio vulnificus; C064778:LuxR autoinducer binding proteins; D020689:Exopeptidases; D013437:Carbon-Sulfur Lyases; C117753:LuxS protein, Bacteria