Normal responses to injury prevent systemic inflammation and can be immunosuppressive.
Munford
R S
RS
Molecular Host Defense Laboratory, Division of Infectious Diseases, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas 75390-9113, USA. Robert.munford@utsouthwestern.edu
Pugin
J
J
D000222:Adaptation, Physiological / Q000502:physiology; D016207:Cytokines / Q000502:physiology; D006801:Humans; D007108:Immune Tolerance / Q000276:immunology; D018836:Inflammation Mediators / Q000502:physiology; D007375:Interleukin-1 / Q000502:physiology; D012772:Shock, Septic / Q000276:immunology*; D018746:Systemic Inflammatory Response Syndrome / Q000276:immunology*; D014409:Tumor Necrosis Factor-alpha / Q000502:physiology
D016207:Cytokines; D018836:Inflammation Mediators; D007375:Interleukin-1; D014409:Tumor Necrosis Factor-alpha