Toll-like receptor signaling and chemokine receptor expression influence the severity of urinary tract infection.
Svanborg
C
C
Department of Microbiology, Immunology and Glycobiology, Institute of Laboratory Medicine, Lund University, S-223 62 Lund, Sweden. Catharina.Svanborg@mig.lu.se
Frendéus
B
B
Godaly
G
G
Hang
L
L
Hedlund
M
M
Wachtler
C
C
D018925:Chemokines / Q000502:physiology; D029721:Drosophila Proteins; D006801:Humans; D008562:Membrane Glycoproteins / Q000502:physiology*; D009504:Neutrophils / Q000502:physiology; D011704:Pyelonephritis / Q000378:metabolism / Q000473:pathology; D011956:Receptors, Cell Surface / Q000502:physiology*; D019707:Receptors, Chemokine / Q000096:biosynthesis / Q000502:physiology*; D023062:Receptors, Interleukin-8A / Q000378:metabolism; D012720:Severity of Illness Index; D015398:Signal Transduction / Q000502:physiology; D051193:Toll-Like Receptors; D014552:Urinary Tract Infections / Q000378:metabolism* / Q000473:pathology
D018925:Chemokines; D029721:Drosophila Proteins; D008562:Membrane Glycoproteins; D011956:Receptors, Cell Surface; D019707:Receptors, Chemokine; D023062:Receptors, Interleukin-8A; D051193:Toll-Like Receptors