[Decrease of the Ca(2+)-dependent K(+)-current by cyclic guanosine monophosphate does not depend on phosphorylation].
Solntseva
E I
EI
Institute of Brian Research, Per. Obukha, 5, 103064, Moscow, Russia.
Bukanova
Iu V
IuV
D000818:Animals; D002118:Calcium / Q000378:metabolism*; D002463:Cell Membrane Permeability; D006152:Cyclic GMP / Q000031:analogs & derivatives* / Q000494:pharmacology / Q000502:physiology*; D003995:Dibutyryl Cyclic GMP / Q000494:pharmacology; D004558:Electric Stimulation; D017952:Ganglia, Invertebrate / Q000187:drug effects / Q000502:physiology*; D066298:In Vitro Techniques; D051036:Large-Conductance Calcium-Activated Potassium Channels; D008564:Membrane Potentials; D010766:Phosphorylation; D015221:Potassium Channels / Q000502:physiology*; D024681:Potassium Channels, Calcium-Activated; D012908:Snails
D051036:Large-Conductance Calcium-Activated Potassium Channels; D015221:Potassium Channels; D024681:Potassium Channels, Calcium-Activated; C016276:8-bromocyclic GMP; D003995:Dibutyryl Cyclic GMP; D006152:Cyclic GMP; D002118:Calcium
Umen'shenie Ca(2+)-zavisimogo K(+)-toka tsiklicheskim guanozinmonofosfatom ne zavisit ot fosforilirovaniia.