Possible cellular mechanism accounting for the cell type-specific vulnerability in Huntington's disease.
Centonze
D
D
Neurological Clinic, Department of Neurosciences, University Tor Vergata and IRCCS S. Lucia Hospital, Rome, Italy.
Giacomini
P
P
Tolu
M
M
Bernardi
G
G
Calabresi
P
P
D015220:Calcium Channels / Q000378:metabolism; D002799:Cholinergic Fibers / Q000378:metabolism / Q000473:pathology; D003342:Corpus Striatum / Q000378:metabolism / Q000473:pathology; D005947:Glucose / Q000172:deficiency; D006801:Humans; D006816:Huntington Disease / Q000378:metabolism* / Q000473:pathology*; D007425:Intracellular Membranes / Q000378:metabolism; D017136:Ion Transport / Q000502:physiology; D009410:Nerve Degeneration / Q000378:metabolism* / Q000473:pathology*; D010100:Oxygen / Q000378:metabolism; D017470:Receptors, Glutamate / Q000378:metabolism; D016194:Receptors, N-Methyl-D-Aspartate / Q000378:metabolism
D015220:Calcium Channels; D017470:Receptors, Glutamate; D016194:Receptors, N-Methyl-D-Aspartate; D005947:Glucose; D010100:Oxygen