K(+) depletion and the progression of hypertensive disease or heart failure. The pathogenic role of diuretic-induced aldosterone secretion.
Laragh
J H
JH
Cardiovascular Center, Weill Medical College of Cornell University, New York, NY, USA.
Sealey
J E
JE
D000450:Aldosterone / Q000378:metabolism*; D000818:Animals; D000959:Antihypertensive Agents / Q000627:therapeutic use*; D001581:Benzothiadiazines; D002986:Clinical Trials as Topic; D004195:Disease Models, Animal; D004232:Diuretics; D006333:Heart Failure / Q000188:drug therapy* / Q000209:etiology; D006801:Humans; D006973:Hypertension / Q000188:drug therapy* / Q000209:etiology; D007008:Hypokalemia / Q000139:chemically induced* / Q000150:complications; D000451:Mineralocorticoid Receptor Antagonists / Q000627:therapeutic use*; D009132:Muscles / Q000378:metabolism; D011188:Potassium / Q000097:blood / Q000378:metabolism* / Q000652:urine; D049993:Sodium Chloride Symporter Inhibitors / Q000009:adverse effects / Q000627:therapeutic use; D013148:Spironolactone / Q000627:therapeutic use*; D016896:Treatment Outcome
D000959:Antihypertensive Agents; D001581:Benzothiadiazines; D004232:Diuretics; D000451:Mineralocorticoid Receptor Antagonists; D049993:Sodium Chloride Symporter Inhibitors; D013148:Spironolactone; D000450:Aldosterone; D011188:Potassium