Increased Ca2+ buffering enhances Ca2+-dependent process.
Zucker
R S
RS
Department of Molecular and Cell Biology, University of California, Berkeley, CA 94720-3200, USA. zucker@socrates.berkeley.edu
D000818:Animals; D002021:Buffers; D002118:Calcium / Q000502:physiology*; D002614:Chelating Agents / Q000494:pharmacology; D004533:Egtazic Acid / Q000031:analogs & derivatives / Q000494:pharmacology; D017729:Presynaptic Terminals / Q000187:drug effects / Q000502:physiology; D009435:Synaptic Transmission / Q000187:drug effects / Q000502:physiology
D002021:Buffers; D002614:Chelating Agents; D004533:Egtazic Acid; C025603:1,2-bis(2-aminophenoxy)ethane-N,N,N',N'-tetraacetic acid; D002118:Calcium