Role of Fas/Fasl in Kupffer cell-dependent deletion of alloantigen activated T cells following liver transplantation.
Sun
Z
Z
Department of Surgery, The Johns Hopkins Medical Institutions, Baltimore, Maryland, USA.
Wada
T
T
Uchikura
K
K
Ceppa
E
E
Klein
A S
AS
D000818:Animals; D017209:Apoptosis / Q000276:immunology*; D003602:Cytotoxicity, Immunologic; D053222:Fas Ligand Protein; D006801:Humans; D019169:Jurkat Cells / Q000276:immunology; D007728:Kupffer Cells / Q000276:immunology* / Q000378:metabolism; D016031:Liver Transplantation / Q000276:immunology*; D008213:Lymphocyte Activation; D008297:Male; D008562:Membrane Glycoproteins / Q000378:metabolism*; D051381:Rats; D011917:Rats, Inbred Lew; D013601:T-Lymphocytes / Q000276:immunology / Q000502:physiology*; D019014:fas Receptor / Q000378:metabolism*
C505661:FASLG protein, human; D053222:Fas Ligand Protein; C505662:Faslg protein, rat; D008562:Membrane Glycoproteins; D019014:fas Receptor