Hypothesis: is a failure to prevent bacteriolysis and the synergy among microbial and host-derived pro-inflammatory agonists the main contributory factors to the pathogenesis of post-infectious sequelae?
Ginsburg
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I
Department of Oral Biology, Hebrew University-Hadassah Faculty of Dental Medicine, Jerusalem, Israel.
D000818:Animals; D000900:Anti-Bacterial Agents / Q000494:pharmacology / Q000627:therapeutic use; D000893:Anti-Inflammatory Agents / Q000008:administration & dosage / Q000627:therapeutic use; D016470:Bacteremia / Q000150:complications / Q000175:diagnosis / Q000188:drug therapy; D001433:Bacteriolysis / Q000187:drug effects; D002473:Cell Wall / Q000187:drug effects / Q000378:metabolism; D003422:Critical Care; D003469:Culture; D004334:Drug Administration Schedule; D004359:Drug Therapy, Combination; D004731:Endotoxins / Q000378:metabolism; D006801:Humans; D018836:Inflammation Mediators / Q000502:physiology*; D008070:Lipopolysaccharides / Q000378:metabolism; D008954:Models, Biological; D010586:Phagocytes / Q000201:enzymology; D010587:Phagocytosis; D018805:Sepsis / Q000188:drug therapy / Q000503:physiopathology* / Q000517:prevention & control
D000900:Anti-Bacterial Agents; D000893:Anti-Inflammatory Agents; D004731:Endotoxins; D018836:Inflammation Mediators; D008070:Lipopolysaccharides