Dystonia and cerebellar atrophy in Cacna1a null mice lacking P/Q calcium channel activity.
Fletcher
C F
CF
Mouse Cancer Genetics Program, National Cancer Institute-FCRDC, Frederick, Maryland 21702, USA. Fletcher@gnf.org
Tottene
A
A
Lennon
V A
VA
Wilson
S M
SM
Dubel
S J
SJ
Paylor
R
R
Hosford
D A
DA
Tessarollo
L
L
McEnery
M W
MW
Pietrobon
D
D
Copeland
N G
NG
Jenkins
N A
NA
D000818:Animals; D002121:Calcium Channel Blockers / Q000494:pharmacology; D015220:Calcium Channels / Q000235:genetics / Q000378:metabolism*; D020864:Calcium Channels, N-Type; D020865:Calcium Channels, P-Type; D020907:Calcium Channels, Q-Type; D002478:Cells, Cultured; D002524:Cerebellar Ataxia / Q000235:genetics / Q000503:physiopathology*; D002531:Cerebellum / Q000166:cytology / Q000187:drug effects / Q000473:pathology* / Q000503:physiopathology; D018390:Gene Targeting; D006801:Humans; D051379:Mice; D008822:Mice, Transgenic; D009553:Nimodipine / Q000494:pharmacology; D018408:Patch-Clamp Techniques; D020866:omega-Conotoxin GVIA / Q000494:pharmacology; D020960:omega-Conotoxins / Q000494:pharmacology
D002121:Calcium Channel Blockers; D015220:Calcium Channels; D020864:Calcium Channels, N-Type; D020865:Calcium Channels, P-Type; D020907:Calcium Channels, Q-Type; D009553:Nimodipine; D020866:omega-Conotoxin GVIA; D020960:omega-Conotoxins; C105656:CACNA1A protein, human; C075851:omega-conotoxin-MVIIC