[A case of suspected pulmonary air embolism in endoscopic neurosurgery].
Sudden reduction in end-tidal PCO2 and SpO2 occurred during the endoscopic third ventriculostomy in a patient with hydrocephalus under general anesthesia. We suspect that it was caused by pulmonary air embolism. A 63-year-old female was scheduled for endoscopic third ventriculostomy under general anesthesia. Endoscopic manipulation caused hemorrhage from chorioid plexus 21 minutes after the procedure was begun, and intraventricular irrigation was performed to achieve hemostasis. In the subsequent 3 minutes, end-tidal PCO2 declined from 26 mmHg to 15 mmHg (PaCO2 39.6 mmHg), and SpO2 declined from 98% to 92% (PaO2 69.2 mmHg). Nitrous oxide was discontinued immediately because pulmonary air embolism was suspected and the oxygen concentration was increased to 100%. At the same time the surgical procedure was discontinued. After 15 minutes, end-tidal PCO2 recovered to 25 mmHg, and SpO2 recovered to 98% (PaO2 136.5 mmHg), and surgery was resumed. The patient recovered from anesthesia. The chest X-p at the end of operation, and pulmonary scintigraphy on the following day revealed no abnormal findings, but brain CT demonstrated a large quantity of air in both lateral ventricles.
Kida
H
H
Department of Anesthesia and Department of Neurosurgery, Suita Municipal Hospital, Suita 564-0082.
Nishikawa
N
N
Ohta
M
M
Miyao
S
S
Takimoto
H
H
D004618:Embolism, Air / Q000209:etiology*; D004724:Endoscopy; D005260:Female; D006801:Humans; D008875:Middle Aged; D009493:Neurosurgery / Q000379:methods*; D011183:Postoperative Complications; D011655:Pulmonary Embolism / Q000209:etiology*