Suppression of the tetrodotoxin-resistant sodium channel (PN3/SNS): a possible new treatment for bladder pain.
Yoshimura
N
N
Department of Pharmacology and Urology, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, USA.
Seki
S
S
Erickson
V
V
Erickson
K
K
Kassotakis
L
L
Novakovic
S
S
Fraser
M O
MO
Chancellor
M B
MB
de Groat
W C
WC
D019342:Acetic Acid; D000818:Animals; D004351:Drug Resistance; D005260:Female; D007202:Indicators and Reagents; D062559:NAV1.8 Voltage-Gated Sodium Channel; D020319:Oligodeoxyribonucleotides, Antisense / Q000627:therapeutic use*; D010146:Pain / Q000139:chemically induced / Q000378:metabolism; D059408:Pain Management; D016760:Proto-Oncogene Proteins c-fos / Q000378:metabolism; D051381:Rats; D015222:Sodium Channels / Q000187:drug effects* / Q000378:metabolism; D013779:Tetrodotoxin / Q000494:pharmacology; D001743:Urinary Bladder / Q000187:drug effects* / Q000294:innervation / Q000378:metabolism; D001745:Urinary Bladder Diseases / Q000139:chemically induced / Q000378:metabolism / Q000628:therapy*
D007202:Indicators and Reagents; D062559:NAV1.8 Voltage-Gated Sodium Channel; D020319:Oligodeoxyribonucleotides, Antisense; D016760:Proto-Oncogene Proteins c-fos; C568319:Scn10a protein, rat; D015222:Sodium Channels; D013779:Tetrodotoxin; D019342:Acetic Acid