Possible mechanisms to explain the absence of hyperkalaemia in Addison's disease.
Gagnon
R F
RF
Renal Division, Montreal General Hospital, McGill University, Montreal, Quebec, Canada.
Halperin
M L
ML
D000224:Addison Disease / Q000097:blood / Q000503:physiopathology* / Q000628:therapy; D000324:Adrenocorticotropic Hormone; D002712:Chlorides / Q000097:blood / Q000652:urine; D003404:Creatinine / Q000097:blood / Q000652:urine; D006706:Homeostasis; D006801:Humans; D006854:Hydrocortisone / Q000031:analogs & derivatives / Q000097:blood / Q000627:therapeutic use*; D006947:Hyperkalemia; D007010:Hyponatremia / Q000175:diagnosis / Q000209:etiology*; D007262:Infusions, Intravenous; D007668:Kidney / Q000503:physiopathology; D008297:Male; D008875:Middle Aged; D008954:Models, Biological; D011188:Potassium / Q000097:blood* / Q000652:urine; D012964:Sodium / Q000097:blood* / Q000652:urine; D012965:Sodium Chloride / Q000008:administration & dosage / Q000627:therapeutic use; D013972:Thyrotropin / Q000097:blood
D002712:Chlorides; D012965:Sodium Chloride; D000324:Adrenocorticotropic Hormone; D013972:Thyrotropin; D012964:Sodium; D003404:Creatinine; C007133:hydrocortisone hemisuccinate; D011188:Potassium; D006854:Hydrocortisone