title: Potassium channels in the human myometrium.
abstract: The contractility of the human uterus is under the fine control of a variety of interacting bioactive agents. During labour, the excitability of the uterus is drastically transformed in comparison with the non-labour state and is manifest at the membrane level via the activity of uterine ion channels. This article reviews the contribution of potassium (K(+)) channels to human uterine excitability. Experimental Physiology (2001) 86.2, 255-264.
authors: Academic Division of Obstetrics and Gynaecology, University of Nottingham, Derby City General Hospital, Uttoxeter New Road, Derby DE22 3NE, UK. raheela.khan@nottingham.ac.uk
authors: R N
authors: RN
authors: Khan
authors: B
authors: B
authors: Matharoo-Ball
authors: S
authors: S
authors: Arulkumaran
authors: M L
authors: ML
authors: Ashford
mesh_terms: D000255:Adenosine Triphosphate / Q000502:physiology; D002118:Calcium / Q000502:physiology; D004594:Electrophysiology; D005260:Female; D006801:Humans; D015640:Ion Channel Gating; D007743:Labor, Obstetric / Q000378:metabolism; D009215:Myometrium / Q000502:physiology*; D015221:Potassium Channels / Q000502:physiology*; D011247:Pregnancy; D014590:Uterine Contraction / Q000502:physiology
chemical_list: D015221:Potassium Channels; D000255:Adenosine Triphosphate; D002118:Calcium