Uptake and hydrolysis of sphingomyelin analogues in Plasmodium falciparum-infected red cells.
Lauer
S A
SA
Department of Microbiology and Immunology, Stanford University, School of Medicine, 94305-5402, Stanford, CA, USA. sabine@telomere.lanl.gov
Chatterjee
S
S
Haldar
K
K
D009327:4-Chloro-7-nitrobenzofurazan / Q000031:analogs & derivatives* / Q000378:metabolism; D000818:Animals; D002518:Ceramides / Q000378:metabolism; D004912:Erythrocytes / Q000469:parasitology*; D006801:Humans; D016778:Malaria, Falciparum / Q000469:parasitology; D010963:Plasmodium falciparum / Q000201:enzymology*; D013108:Sphingomyelin Phosphodiesterase / Q000378:metabolism*; D013109:Sphingomyelins / Q000737:chemistry* / Q000378:metabolism*
D002518:Ceramides; D013109:Sphingomyelins; C037974:N-(7-(4-nitrobenzo-2-oxa-1,3-diazole))-6-aminocaproyl sphingosine; C081708:N-(N-(7-nitro-2,1,3-benzoxadiazol-4-yl)-epsilon-aminohexanoyl)sphingosylphosphorylcholine; D013108:Sphingomyelin Phosphodiesterase; D009327:4-Chloro-7-nitrobenzofurazan