Varicella-zoster virus: molecular virology and virus-host interactions.
Cosmid-based mutagenesis and methods to examine varicella-zoster virus (VZV) tropism for differentiated human cells in vivo provide new information about molecular mechanisms of VZV infection. How specific VZV gene products contribute to viral replication has been further defined, and effects of VZV on expression of cellular genes have been demonstrated.
Arvin
A M
AM
G-312, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, CA 94305, USA. aarvin@stanford.edu
D002460:Cell Line; D002478:Cells, Cultured; D002644:Chickenpox / Q000821:virology*; D003360:Cosmids / Q000235:genetics; D006562:Herpes Zoster / Q000821:virology*; D014645:Herpesvirus 3, Human / Q000235:genetics* / Q000472:pathogenicity* / Q000502:physiology; D006801:Humans; D016296:Mutagenesis; D014774:Virulence / Q000235:genetics; D014779:Virus Replication