[Lectin-induced platelet aggregation inpatients with unstable angina pectoris treated with anti-aggregation agents].
Lectin-induced platelet aggregation was studied in patients with unstable angina pectoris treated with tiklid and aspirin. ADP, PHA-P, WGA, Con A served as the aggregation inductors. In unstable angina pectoris lectin-induced platelet aggregation was enhanced. Changes in this aggregation in response to treatment with antiaggregants with different mechanisms of action are demonstrated.
Voskoboĭ
I V
IV
Rebrov
A P
AP
Kirichuk
V F
VF
Pozin
P Ia
PIa
D000249:Adenosine Monophosphate / Q000009:adverse effects*; D000789:Angina, Unstable / Q000175:diagnosis / Q000188:drug therapy*; D001792:Blood Platelets / Q000378:metabolism*; D005260:Female; D006801:Humans; D037102:Lectins / Q000009:adverse effects*; D008297:Male; D008875:Middle Aged; D010835:Phytohemagglutinins / Q000009:adverse effects*; D010975:Platelet Aggregation Inhibitors / Q000494:pharmacology / Q000627:therapeutic use*; D013927:Thrombosis / Q000139:chemically induced*; D013988:Ticlopidine / Q000494:pharmacology / Q000627:therapeutic use*
D037102:Lectins; D010835:Phytohemagglutinins; D010975:Platelet Aggregation Inhibitors; C028784:phytohemagglutinin-P; D000249:Adenosine Monophosphate; D013988:Ticlopidine