Predisposing genes, high-risk environments and coronary artery disease: LPL and fibrinogen as examples.
Humphries
S E
SE
Centre for Cardiovascular Genetics, Department of Medicine, UCLMS, Rayne Institute, University Street, London, UK. rmhaseh@ucl.ac.uk
Montgomery
H E
HE
Talmud
P J
PJ
D003324:Coronary Artery Disease / Q000097:blood* / Q000209:etiology / Q000235:genetics* / Q000517:prevention & control; D004777:Environment; D015444:Exercise / Q000502:physiology; D005340:Fibrinogen / Q000235:genetics* / Q000378:metabolism; D020022:Genetic Predisposition to Disease; D006706:Homeostasis; D006801:Humans; D008019:Life Style; D008071:Lipoprotein Lipase / Q000097:blood / Q000235:genetics*; D009765:Obesity / Q000150:complications / Q000235:genetics / Q000378:metabolism; D012307:Risk Factors; D014280:Triglycerides / Q000097:blood
D014280:Triglycerides; D005340:Fibrinogen; D008071:Lipoprotein Lipase