title: [Protein metabolism impairment in long-term crush syndrome].
abstract: Experiments on Wistar rat males modelled crush syndrome (CS). Stability of blood protein composition in rats undergoing CS decompression was found. This reflects activation of protein synthesis in the defense systems of the body due to stress reaction to CS. It seems that the lymphatic system takes a detoxicating function which is essential in conditions when the same function of the liver in CS weakens. The conclusion is made on stressor depletion in CS as a typical pathological process rapidly realizing main links of alimentary dystrophy.
authors.forename: A V
authors.initials: AV
authors.lastname: Efremov
authors.forename: A R
authors.initials: AR
authors.lastname: Antonov
authors.forename: Iu V
authors.initials: IuV
authors.lastname: Nacharov
authors.forename: E N
authors.initials: EN
authors.lastname: Samsonova
authors.forename: V Iu
authors.initials: VIu
authors.lastname: Radustov
mesh_terms: D000418:Albumins / Q000378:metabolism; D000818:Animals; D003404:Creatinine / Q000097:blood / Q000378:metabolism; D003444:Crush Syndrome / Q000097:blood / Q000378:metabolism*; D008208:Lymphatic System / Q000378:metabolism; D008297:Male; D011506:Proteins / Q000378:metabolism*; D051381:Rats; D017208:Rats, Wistar; D014508:Urea / Q000097:blood / Q000378:metabolism; D014527:Uric Acid / Q000097:blood / Q000378:metabolism
chemical_list: D000418:Albumins; D011506:Proteins; D014527:Uric Acid; D014508:Urea; D003404:Creatinine