[Antioxidant effect of low intensity laser infrared irradiation].
Effects of low-intensive infrared laser (890 nm) exposure on lipid peroxidation (LPO) and antioxidant system (AOS) activity in the membrane fraction of hepatocyte endoplasmatic reticulum (microsomal fraction of the liver) were studied in a physiologically balanced LPO/AOS system (intact animals) and under conditions of LPO initiation by immobilization cold stress (animals with erosions and ulcers of the gastric mucosa after Shay). Antioxidant effect in laser exposure of the liver depends on the frequency of pulses, is mediated by laser effect on hepatocyte membrane structures, and manifests by recovery of LPO/AOS system imbalance induced by disease.
Zolotareva
T A
TA
Oleshko
A Ia
AIa
Oleshko
T I
TI
D000818:Animals; D002374:Catalase / Q000378:metabolism; D004721:Endoplasmic Reticulum / Q000378:metabolism / Q000528:radiation effects*; D005979:Glutathione Peroxidase / Q000378:metabolism; D005980:Glutathione Reductase / Q000378:metabolism; D022781:Hepatocytes / Q000378:metabolism / Q000528:radiation effects*; D007259:Infrared Rays; D007425:Intracellular Membranes / Q000378:metabolism / Q000528:radiation effects; D007834:Lasers; D015227:Lipid Peroxidation; D010437:Peptic Ulcer / Q000209:etiology / Q000378:metabolism; D051381:Rats; D017208:Rats, Wistar; D013315:Stress, Psychological / Q000150:complications; D013482:Superoxide Dismutase / Q000378:metabolism
D002374:Catalase; D005979:Glutathione Peroxidase; D013482:Superoxide Dismutase; D005980:Glutathione Reductase