Disturbance of endothelial barrier function by bacterial toxins and atherogenic mediators: a role for Rho/Rho kinase.
Aepfelbacher
M
M
Max von Pettenkofer-Institut für Medizinische Mikrobiologie, München, Germany. aepfelbacher@m3401.mpk.med.uni-muenchen.de
Essler
M
M
D000199:Actins / Q000378:metabolism; D001161:Arteriosclerosis / Q000209:etiology; D001427:Bacterial Toxins / Q000494:pharmacology*; D004730:Endothelium, Vascular / Q000166:cytology / Q000187:drug effects / Q000502:physiology*; D004789:Enzyme Activation; D006801:Humans; D007365:Intercellular Junctions / Q000378:metabolism; D047908:Intracellular Signaling Peptides and Proteins; D009218:Myosins / Q000378:metabolism; D010539:Permeability; D010766:Phosphorylation; D017346:Protein Serine-Threonine Kinases / Q000378:metabolism*; D015398:Signal Transduction; D020741:rho GTP-Binding Proteins / Q000378:metabolism*; D054460:rho-Associated Kinases
D000199:Actins; D001427:Bacterial Toxins; D047908:Intracellular Signaling Peptides and Proteins; D017346:Protein Serine-Threonine Kinases; D054460:rho-Associated Kinases; D009218:Myosins; D020741:rho GTP-Binding Proteins