Development of type 1 diabetes despite severe hereditary B-cell deficiency.
Martin
S
S
German Diabetes Center, German Diabetes Research Institute, Heinrich Heine University, Düsseldorf. martin@dfi.uni-duesseldorf.de
Wolf-Eichbaum
D
D
Duinkerken
G
G
Scherbaum
W A
WA
Kolb
H
H
Noordzij
J G
JG
Roep
B O
BO
D000293:Adolescent; D000077329:Agammaglobulinaemia Tyrosine Kinase; D000361:Agammaglobulinemia / Q000150:complications* / Q000235:genetics; D015703:Antigens, CD / Q000032:analysis; D001323:Autoantibodies / Q000097:blood*; D001402:B-Lymphocytes / Q000502:physiology; D002455:Cell Division; D016207:Cytokines / Q000097:blood; D003922:Diabetes Mellitus, Type 1 / Q000209:etiology / Q000235:genetics / Q000276:immunology*; D008040:Genetic Linkage; D006801:Humans; D008297:Male; D011505:Protein-Tyrosine Kinases / Q000235:genetics; D017422:Sequence Analysis, DNA; D013601:T-Lymphocytes / Q000166:cytology / Q000276:immunology; D014960:X Chromosome
D015703:Antigens, CD; D001323:Autoantibodies; D016207:Cytokines; D011505:Protein-Tyrosine Kinases; D000077329:Agammaglobulinaemia Tyrosine Kinase