Chlamydia pneumoniae exacerbates aortic inflammatory foci caused by murine cytomegalovirus infection in normocholesterolemic mice.
Inflammatory foci induced by murine cytomegalovirus infection in normocholesterolemic mice were present temporarily in the aortic wall, but some of these foci developed into advanced lesions that persisted late after infection. The early foci induced by virus infection were significantly exacerbated following a single inoculation with Chlamydia pneumoniae.
Burian
K
K
Department of Medical Microbiology, Szeged, Hungary.
Berencsi
K
K
Endresz
V
V
Gyulai
Z
Z
Valyi-Nagy
T
T
Valyi-Nagy
I
I
Bakay
M
M
Geng
Y
Y
Virok
D
D
Kari
L
L
Hajnal-Papp
R
R
Trinchieri
G
G
Gonczol
E
E
D000818:Animals; D001025:Aortitis / Q000382:microbiology / Q000473:pathology / Q000821:virology*; D001161:Arteriosclerosis / Q000276:immunology / Q000473:pathology; D023521:Chlamydophila Infections / Q000276:immunology* / Q000473:pathology / Q000821:virology; D016993:Chlamydophila pneumoniae; D002784:Cholesterol / Q000097:blood; D003586:Cytomegalovirus Infections / Q000276:immunology* / Q000382:microbiology / Q000473:pathology; D008297:Male; D051379:Mice; D008807:Mice, Inbred BALB C; D018410:Pneumonia, Bacterial / Q000276:immunology* / Q000473:pathology / Q000821:virology
D002784:Cholesterol