title: [Hyperparathyroidism secondary to anticonvulsant treatment].
abstract: Treatment with some anticonvulsants is known to lead to liver enzyme induction. Two cases suggest that this interaction is also the mechanism behind anticonvulsant osteomalacia, which is characterised by hypocalcaemia, elevated alkaline phosphatase, and low 25(OH) cholecalciferol.
authors.affiliation: Amtssygehuset Roskilde, medicinsk afdeling.
authors.forename: R S
authors.initials: RS
authors.lastname: Andersen
authors.forename: N
authors.initials: N
authors.lastname: Valeur
mesh_terms: D000469:Alkaline Phosphatase / Q000378:metabolism; D000927:Anticonvulsants / Q000009:adverse effects*; D002112:Calcifediol / Q000378:metabolism; D002220:Carbamazepine / Q000009:adverse effects*; D005260:Female; D006801:Humans; D006962:Hyperparathyroidism, Secondary / Q000139:chemically induced* / Q000378:metabolism; D006996:Hypocalcemia / Q000139:chemically induced; D008875:Middle Aged; D010018:Osteomalacia / Q000139:chemically induced
chemical_list: D000927:Anticonvulsants; D002220:Carbamazepine; D000469:Alkaline Phosphatase; D002112:Calcifediol